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Publications by S. O'Shea

BCL-X Is Expressed in Embryonic and Postnatal Neural Tissues and Functions to Prevent Neuronal Cell Death.

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

English

Related publications

Uncoupling Proteins Prevent Glucose-Induced Neuronal Oxidative Stress and Programmed Cell Death

Internal Medicine

English

P53 Deficiency Fails to Prevent Increased Programmed Cell Death in the BCL-Xl-Deficient Nervous System

Cell Death and Differentiation

Molecular Biology

English

Polyglutamine Disease and Neuronal Cell Death

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

English

Fgl2deficiency Causes Neonatal Death and Cardiac Dysfunction During Embryonic and Postnatal Development in Mice

Physiological Genomics

English

BCL-Xl Is Qualitatively Different From and Ten Times More Effective Than BCL-2 When Expressed in a Breast Cancer Cell Line

Cancer Research

English

Apoptosis Repressor Genes BCL-2 and BCL-X-Long Are Expressed in the Rat Brain Following Global Ischemia

Journal of Cerebral Blood Flow and Metabolism

Cardiovascular Medicine

English

BCL-2 Protein as a Marker of Neuronal Immaturity in Postnatal Primate Brain

Journal of Neuroscience

English

Growth Factors Prevent Changes in BCL-2 and Bax Expression and Neuronal Apoptosis Induced by Nitric Oxide

Cell Death and Differentiation

Molecular Biology

English

Hepatocyte Nuclear Factor 3/Fork Head Homolog 11 Is Expressed in Proliferating Epithelial and Mesenchymal Cells of Embryonic and Adult Tissues.

Molecular and Cellular Biology

Molecular Biology

English