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Publications by Saeed Elojeimy

Acid Ceramidase but Not Acid Sphingomyelinase Is Required for Tumor Necrosis Factor-Α-Induced PGE2Production

Journal of Biological Chemistry

English

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Glutathione Regulation of Neutral Sphingomyelinase in Tumor Necrosis Factor-Α-Induced Cell Death

Journal of Biological Chemistry

English

NF-κB Activation Is Required for Human Endothelial Survival During Exposure to Tumor Necrosis Factor-Α but Not to Interleukin-1β or Lipopolysaccharide

Journal of Biological Chemistry

English

Tumor Necrosis Factor Α-Induced Inflammation Is Increased but Apoptosis Is Inhibited by Common Food Additive Carrageenan

Journal of Biological Chemistry

English

Host Acid Sphingomyelinase Regulates Microvascular Function Not Tumor Immunity

Cancer Research

Oncology

2004

English

Oxidatively Truncated Phospholipids Are Required Agents of Tumor Necrosis Factor Α (TNFα)-induced Apoptosis

Journal of Biological Chemistry

English

Ursolic Acid Enhances Cyclooxygenases and Tumor Necrosis Factor-Α Expression in Mouse Skin

Bioscience, Biotechnology and Biochemistry

English

Protein Tyrosine Kinase P56lckIs Required for Ceramide-Induced but Not Tumor Necrosis Factor-Induced Activation of NF-κB, AP-1, JNK, and Apoptosis

Journal of Biological Chemistry

English

Tumor Necrosis Factor-Α–induced Protein 3 (TNFAIP3; A20)

Science-Business eXchange

2010

English

LIGHT, a Member of the Tumor Necrosis Factor Ligand Superfamily, Prevents Tumor Necrosis Factor-Α-Mediated Human Primary Hepatocyte Apoptosis, but Not Fas-Mediated Apoptosis

Journal of Biological Chemistry

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Saeed Elojeimy

Acid Ceramidase but Not Acid Sphingomyelinase Is Required for Tumor Necrosis Factor-Α-Induced PGE2Production

Related publications

Glutathione Regulation of Neutral Sphingomyelinase in Tumor Necrosis Factor-Α-Induced Cell Death

NF-κB Activation Is Required for Human Endothelial Survival During Exposure to Tumor Necrosis Factor-Α but Not to Interleukin-1β or Lipopolysaccharide

Tumor Necrosis Factor Α-Induced Inflammation Is Increased but Apoptosis Is Inhibited by Common Food Additive Carrageenan

Host Acid Sphingomyelinase Regulates Microvascular Function Not Tumor Immunity

Oxidatively Truncated Phospholipids Are Required Agents of Tumor Necrosis Factor Α (TNFα)-induced Apoptosis

Ursolic Acid Enhances Cyclooxygenases and Tumor Necrosis Factor-Α Expression in Mouse Skin

Protein Tyrosine Kinase P56lckIs Required for Ceramide-Induced but Not Tumor Necrosis Factor-Induced Activation of NF-κB, AP-1, JNK, and Apoptosis

Tumor Necrosis Factor-Α–induced Protein 3 (TNFAIP3; A20)

LIGHT, a Member of the Tumor Necrosis Factor Ligand Superfamily, Prevents Tumor Necrosis Factor-Α-Mediated Human Primary Hepatocyte Apoptosis, but Not Fas-Mediated Apoptosis