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Publications by Salama A. Salama

Erratum to “Overhydroxylation of Lysine of Collagen Increases Uterine Fibroids Proliferation: Roles of Lysyl Hydroxylases, Lysyl Oxidases, and Matrix Metalloproteinases”

BioMed Research International

English

Memy I: A Novel Murine Model for Uterine Leiomyoma Using Adenovirus-Enhanced Human Fibroid Explants in Severe Combined Immune Deficiency Mice

American Journal of Obstetrics and Gynecology

Gynecology

Obstetrics

2008

English

Related publications

Lysyl Oxidases Regulate Fibrillar Collagen Remodelling in Idiopathic Pulmonary Fibrosis

DMM Disease Models and Mechanisms

English

Figure 5: Multiple Alignment of Archaeal Lysyl Oxidases.

English

Differential Cleavage of Lysyl Oxidase by the Metalloproteinases BMP1 and ADAMTS2/14 Regulates Collagen Binding Through a Tyrosine Sulfate Domain

Journal of Biological Chemistry

English

The Lysyl Oxidases LOX and LOXL2 Are Necessary and Sufficient to Repress E-Cadherin in Hypoxia

Journal of Biological Chemistry

English

Proteolytic Processing of Lysyl Oxidase Like-2 in the Extracellular Matrix Is Required for Crosslinking of Basement Membrane Collagen IV

Journal of Biological Chemistry

English

Lysyl Hydroxylase 3 Is a Multifunctional Protein Possessing Collagen Glucosyltransferase Activity

Journal of Biological Chemistry

English

The Extracellular Matrix Contributes to Mechanotransduction in Uterine Fibroids

Obstetrics and Gynecology International

Gynecology

Obstetrics

2014

English

Developing Functional Musculoskeletal Tissues Through Hypoxia and Lysyl Oxidase-Induced Collagen Cross-Linking

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

2014

English

Lysyl Oxidase Activity Is Required for Ordered Collagen Fibrillogenesis by Tendon Cells

Journal of Biological Chemistry

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Salama A. Salama

Erratum to “Overhydroxylation of Lysine of Collagen Increases Uterine Fibroids Proliferation: Roles of Lysyl Hydroxylases, Lysyl Oxidases, and Matrix Metalloproteinases”

Memy I: A Novel Murine Model for Uterine Leiomyoma Using Adenovirus-Enhanced Human Fibroid Explants in Severe Combined Immune Deficiency Mice

Related publications

Lysyl Oxidases Regulate Fibrillar Collagen Remodelling in Idiopathic Pulmonary Fibrosis

Figure 5: Multiple Alignment of Archaeal Lysyl Oxidases.

Differential Cleavage of Lysyl Oxidase by the Metalloproteinases BMP1 and ADAMTS2/14 Regulates Collagen Binding Through a Tyrosine Sulfate Domain

The Lysyl Oxidases LOX and LOXL2 Are Necessary and Sufficient to Repress E-Cadherin in Hypoxia

Proteolytic Processing of Lysyl Oxidase Like-2 in the Extracellular Matrix Is Required for Crosslinking of Basement Membrane Collagen IV

Lysyl Hydroxylase 3 Is a Multifunctional Protein Possessing Collagen Glucosyltransferase Activity

The Extracellular Matrix Contributes to Mechanotransduction in Uterine Fibroids

Developing Functional Musculoskeletal Tissues Through Hypoxia and Lysyl Oxidase-Induced Collagen Cross-Linking

Lysyl Oxidase Activity Is Required for Ordered Collagen Fibrillogenesis by Tendon Cells