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Publications by Salvador J. Diaz-Cano

Increased Induction of Osteopetrosis, but Unaltered Lymphomagenicity, by Murine Leukemia Virus SL3-3 After Mutation of a Nuclear Factor 1 Site in the Enhancer

Journal of Virology

English

Related publications

Second-Site Proviral Enhancer Alterations in Lymphomas Induced by Enhancer Mutants of SL3-3 Murine Leukemia Virus: Negative Effect of Nuclear Factor 1 Binding Site.

English

Increased Lymphomagenicity and Restored Disease Specificity of AML1 Site (Core) Mutant SL3-3 Murine Leukemia Virus by a Second-Site Enhancer Variant Evolved in Vivo.

English

Stability of AML1 (Core) Site Enhancer Mutations in T Lymphomas Induced by Attenuated SL3-3 Murine Leukemia Virus Mutants.

English

Mutation of All Runx (AML1/Core) Sites in the Enhancer of T-Lymphomagenic SL3-3 Murine Leukemia Virus Unmasks a Significant Potential for Myeloid Leukemia Induction and Favors Enhancer Evolution Toward Induction of Other Disease Patterns

English

Tissue Distribution and Timing of Appearance of Polytropic Envelope Recombinants During Infection With SL3-3 Murine Leukemia Virus or Its Weakly Pathogenic SL3 Myb5 Mutant

English

Novel Type of Hepatitis B Virus Mutation: Replacement Mutation Involving a Hepatocyte Nuclear Factor 1 Binding Site Tandem Repeat in Chronic Hepatitis B Virus Genotype E

English

Herpes Simplex Virus Type 1 Promoted the Possibility of Leukemia Caused by L6565 Murine Leukemia Virus

The Open Veterinary Science Journal

2008

English

Lymphomagenicity of Recombinant Mink Cell Focus-Inducing Murine Leukemia Viruses

Journal of Experimental Medicine

English

The E47 Transcription Factor Binds to the Enhancer Sequences of Recombinant Murine Leukemia Viruses and Influences Enhancer Function.

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Salvador J. Diaz-Cano

Increased Induction of Osteopetrosis, but Unaltered Lymphomagenicity, by Murine Leukemia Virus SL3-3 After Mutation of a Nuclear Factor 1 Site in the Enhancer

Related publications

Second-Site Proviral Enhancer Alterations in Lymphomas Induced by Enhancer Mutants of SL3-3 Murine Leukemia Virus: Negative Effect of Nuclear Factor 1 Binding Site.

Increased Lymphomagenicity and Restored Disease Specificity of AML1 Site (Core) Mutant SL3-3 Murine Leukemia Virus by a Second-Site Enhancer Variant Evolved in Vivo.

Stability of AML1 (Core) Site Enhancer Mutations in T Lymphomas Induced by Attenuated SL3-3 Murine Leukemia Virus Mutants.

Mutation of All Runx (AML1/Core) Sites in the Enhancer of T-Lymphomagenic SL3-3 Murine Leukemia Virus Unmasks a Significant Potential for Myeloid Leukemia Induction and Favors Enhancer Evolution Toward Induction of Other Disease Patterns

Tissue Distribution and Timing of Appearance of Polytropic Envelope Recombinants During Infection With SL3-3 Murine Leukemia Virus or Its Weakly Pathogenic SL3 Myb5 Mutant

Novel Type of Hepatitis B Virus Mutation: Replacement Mutation Involving a Hepatocyte Nuclear Factor 1 Binding Site Tandem Repeat in Chronic Hepatitis B Virus Genotype E

Herpes Simplex Virus Type 1 Promoted the Possibility of Leukemia Caused by L6565 Murine Leukemia Virus

Lymphomagenicity of Recombinant Mink Cell Focus-Inducing Murine Leukemia Viruses

The E47 Transcription Factor Binds to the Enhancer Sequences of Recombinant Murine Leukemia Viruses and Influences Enhancer Function.