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Publications by Sebastián Pons

Insulin-Induced Up-Regulated Uncoupling Protein-1 Expression Is Mediated by Insulin Receptor Substrate 1 Through the Phosphatidylinositol 3-Kinase/Akt Signaling Pathway in Fetal Brown Adipocytes

Journal of Biological Chemistry

English

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Modulation of Insulin Receptor Substrate-1 Tyrosine Phosphorylation by an Akt/Phosphatidylinositol 3-Kinase Pathway

Journal of Biological Chemistry

English

Shiga Toxin 1-Induced Proinflammatory Cytokine Production Is Regulated by the Phosphatidylinositol 3-Kinase/Akt/Mammalian Target of Rapamycin Signaling Pathway

Infection and Immunity

English

Insulin-Like Growth Factor-1 Receptor Activation Inhibits Oxidized LDL-Induced Cytochrome C Release and Apoptosis via the Phosphatidylinositol 3 Kinase/Akt Signaling Pathway

Arteriosclerosis, Thrombosis, and Vascular Biology

Cardiovascular Medicine

Cardiology

2003

English

S-Nitrosation of the Insulin Receptor, Insulin Receptor Substrate 1, and Protein Kinase B/Akt: A Novel Mechanism of Insulin Resistance

English

Tumor Necrosis Factor Promotes Phosphorylation and Binding of Insulin Receptor Substrate 1 to Phosphatidylinositol 3-Kinase in 3t3-L1 Adipocytes

Journal of Biological Chemistry

English

Prolactin Activates Tyrosyl Phosphorylation of Insulin Receptor Substrate 1 and Phosphatidylinositol-3-Oh Kinase

Journal of Biological Chemistry

English

NCS-1 Inhibits Insulin-Stimulated GLUT4 Translocation in 3T3L1 Adipocytes Through a Phosphatidylinositol 4-Kinase-Dependent Pathway

Journal of Biological Chemistry

English

Insulin and Insulin-Like Growth Factor I Up-Regulate GLUT4 Gene Expression in Fetal Brown Adipocytes, in a Phosphoinositide 3-Kinase-Dependent Manner

English

Phosphatidylinositol 3′-Kinase Associates With an Insulin Receptor Substrate-1 Serine Kinase Distinct From Its Intrinsic Serine Kinase

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Sebastián Pons

Insulin-Induced Up-Regulated Uncoupling Protein-1 Expression Is Mediated by Insulin Receptor Substrate 1 Through the Phosphatidylinositol 3-Kinase/Akt Signaling Pathway in Fetal Brown Adipocytes

Related publications

Modulation of Insulin Receptor Substrate-1 Tyrosine Phosphorylation by an Akt/Phosphatidylinositol 3-Kinase Pathway

Shiga Toxin 1-Induced Proinflammatory Cytokine Production Is Regulated by the Phosphatidylinositol 3-Kinase/Akt/Mammalian Target of Rapamycin Signaling Pathway

Insulin-Like Growth Factor-1 Receptor Activation Inhibits Oxidized LDL-Induced Cytochrome C Release and Apoptosis via the Phosphatidylinositol 3 Kinase/Akt Signaling Pathway

S-Nitrosation of the Insulin Receptor, Insulin Receptor Substrate 1, and Protein Kinase B/Akt: A Novel Mechanism of Insulin Resistance

Tumor Necrosis Factor Promotes Phosphorylation and Binding of Insulin Receptor Substrate 1 to Phosphatidylinositol 3-Kinase in 3t3-L1 Adipocytes

Prolactin Activates Tyrosyl Phosphorylation of Insulin Receptor Substrate 1 and Phosphatidylinositol-3-Oh Kinase

NCS-1 Inhibits Insulin-Stimulated GLUT4 Translocation in 3T3L1 Adipocytes Through a Phosphatidylinositol 4-Kinase-Dependent Pathway

Insulin and Insulin-Like Growth Factor I Up-Regulate GLUT4 Gene Expression in Fetal Brown Adipocytes, in a Phosphoinositide 3-Kinase-Dependent Manner

Phosphatidylinositol 3′-Kinase Associates With an Insulin Receptor Substrate-1 Serine Kinase Distinct From Its Intrinsic Serine Kinase