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Publications by Shu-ichi Ueda

PTEN/Akt Signaling Through Epidermal Growth Factor Receptor Is Prerequisite for Angiogenesis by Hepatocellular Carcinoma Cells That Is Susceptible to Inhibition by Gefitinib

Cancer Research

Oncology

2006

English

Lead Generation of Anti-Allergic Agent ; Suplatast Tosilate

Chem-Bio Informatics Journal

Biochemistry

2001

English

Related publications

Fibroblast Growth Factor Signaling Pathway in Endothelial Cells Is Activated by BMPER to Promote Angiogenesis

Arteriosclerosis, Thrombosis, and Vascular Biology

Cardiovascular Medicine

Cardiology

2015

English

JARID2 Promotes Invasion and Metastasis of Hepatocellular Carcinoma by Facilitating Epithelial-Mesenchymal Transition Through PTEN/AKT Signaling

Oncotarget

Oncology

2016

English

Growth and Motility Inhibition of Breast Cancer Cells by Epidermal Growth Factor Receptor Degradation Is Correlated With Inactivation of Cdc42

Cancer Research

Oncology

2006

English

Nuclear Translocation of Epidermal Growth Factor Receptor by Akt-Dependent Phosphorylation Enhances Breast Cancer-Resistant Protein Expression in Gefitinib-Resistant Cells

Journal of Biological Chemistry

English

Activation of Rho Is Required for Ligand-Independent Oncogenic Signaling by a Mutant Epidermal Growth Factor Receptor

Journal of Biological Chemistry

English

Inhibition of VEGF165/VEGFR2-dependent Signaling by LECT2 Suppresses Hepatocellular Carcinoma Angiogenesis

Scientific Reports

Multidisciplinary

2016

English

Bradykinin-Induced Growth Inhibition of Normal Rat Kidney (NRK) Cells Is Paralleled by a Decrease in Epidermal-Growth-Factor Receptor Expression

English

Differential Epidermal Growth Factor Receptor Signaling Regulates Anchorage-Independent Growth by Modulation of the PI3K/AKT Pathway

English

Pathological Angiogenesis Is Induced by Sustained Akt Signaling and Inhibited by Rapamycin

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Shu-ichi Ueda

PTEN/Akt Signaling Through Epidermal Growth Factor Receptor Is Prerequisite for Angiogenesis by Hepatocellular Carcinoma Cells That Is Susceptible to Inhibition by Gefitinib

Lead Generation of Anti-Allergic Agent ; Suplatast Tosilate

Related publications

Fibroblast Growth Factor Signaling Pathway in Endothelial Cells Is Activated by BMPER to Promote Angiogenesis

JARID2 Promotes Invasion and Metastasis of Hepatocellular Carcinoma by Facilitating Epithelial-Mesenchymal Transition Through PTEN/AKT Signaling

Growth and Motility Inhibition of Breast Cancer Cells by Epidermal Growth Factor Receptor Degradation Is Correlated With Inactivation of Cdc42

Nuclear Translocation of Epidermal Growth Factor Receptor by Akt-Dependent Phosphorylation Enhances Breast Cancer-Resistant Protein Expression in Gefitinib-Resistant Cells

Activation of Rho Is Required for Ligand-Independent Oncogenic Signaling by a Mutant Epidermal Growth Factor Receptor

Inhibition of VEGF165/VEGFR2-dependent Signaling by LECT2 Suppresses Hepatocellular Carcinoma Angiogenesis

Bradykinin-Induced Growth Inhibition of Normal Rat Kidney (NRK) Cells Is Paralleled by a Decrease in Epidermal-Growth-Factor Receptor Expression

Differential Epidermal Growth Factor Receptor Signaling Regulates Anchorage-Independent Growth by Modulation of the PI3K/AKT Pathway

Pathological Angiogenesis Is Induced by Sustained Akt Signaling and Inhibited by Rapamycin