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Publications by Swenja Kröller-Schön
Angiotensin II-Induced Vascular Dysfunction Depends on Interferon- -Driven Immune Cell Recruitment and Mutual Activation of Monocytes and NK-Cells
Arteriosclerosis, Thrombosis, and Vascular Biology
Cardiovascular Medicine
Cardiology
Peroxisome Proliferator-Activated Receptor , Coactivator 1 Deletion Induces Angiotensin II-Associated Vascular Dysfunction by Increasing Mitochondrial Oxidative Stress and Vascular Inflammation
Arteriosclerosis, Thrombosis, and Vascular Biology
Cardiovascular Medicine
Cardiology
Related publications
Hypoxia Inducible Factor 1α in Vascular Smooth Muscle Cells Promotes Angiotensin II-induced Vascular Remodeling via Activation of CCL7-mediated Macrophage Recruitment
Cell Death and Disease
Molecular Neuroscience
Immunology
Cell Biology
Cancer Research
Cellular
Medicine
Notch Activation Mediates Angiotensin II-induced Vascular Remodeling by Promoting the Proliferation and Migration of Vascular Smooth Muscle Cells
Hypertension Research
Internal Medicine
Cardiovascular Medicine
Physiology
Cardiology
Activation of MAPKs by Angiotensin II in Vascular Smooth Muscle Cells
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Th1 Type Immune Responses to Porphyromonas Gingivalis Antigens Exacerbate Angiotensin II Dependent Hypertension and Vascular Dysfunction.
British Journal of Pharmacology
Pharmacology
MicroRNA and mRNA Analysis of Angiotensin II‑induced Renal Artery Endothelial Cell Dysfunction
Experimental and Therapeutic Medicine
Medicine
Cancer Research
Immunology
Microbiology
Rosiglitzone Suppresses Angiotensin II-Induced Production of KLF5 and Cell Proliferation in Rat Vascular Smooth Muscle Cells
PLoS ONE
Multidisciplinary
Immune Complexes (IC) Down-Regulate the Basal and Interferon-Gamma-Induced Expression of MHC Class II on Human Monocytes
Clinical and Experimental Immunology
Allergy
Immunology
Aldosterone Potentiates Angiotensin II-Induced Signaling in Vascular Smooth Muscle Cells
Circulation
Cardiovascular Medicine
Physiology
Cardiology
Soluble Fc-Disabled Herpes Virus Entry Mediator Augments Activation and Cytotoxicity of NK Cells by Promoting Cross-Talk Between NK Cells and Monocytes
Journal of Immunology
Allergy
Immunology
