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Publications by T. J. Pullen

Comment On: Schuit Et Al. -Cell-Specific Gene Repression: A Mechanism to Protect Against Inappropriate or Maladjusted Insulin Secretion? Diabetes 2012;61:969-975

Diabetes

English

Related publications

Response to Comment On: Schuit Et Al. -Cell-Specific Gene Repression: A Mechanism to Protect Against Inappropriate or Maladjusted Insulin Secretion? Diabetes 2012;61:969-975

English

Comment On: Turban Et Al. Optimal Elevation of -Cell 11 -Hydroxysteroid Dehydrogenase Type 1 Is a Compensatory Mechanism That Prevents High-Fat Diet-Induced -Cell Failure. Diabetes 2012;61:642-652

English

Comment on Lee Et Al. Diabetes 2015;64:2836–2846. Comment on Roberts Et Al. Diabetes 2015;64:471–484

English

Response to Comment On: Marquez Et Al. Low-Frequency Variants in HMGA1 Are Not Associated With Type 2 Diabetes Risk. Diabetes 2012;61:524-530

English

Response to Comment on Sims Et Al. Proinsulin Secretion Is a Persistent Feature of Type 1 Diabetes. Diabetes Care 2019;42:258–264

English

Comment On: Teeuwisse Et Al. Short-Term Caloric Restriction Normalizes Hypothalamic Neuronal Responsiveness to Glucose Ingestion in Patients With Type 2 Diabetes. Diabetes 2012;61:3255-3259

English

Comment on Sims Et Al. Proinsulin Secretion Is a Persistent Feature of Type 1 Diabetes. Diabetes Care 2019;42:258–264

English

Response to Comment On: Tam Et Al. Defining Insulin Resistance From Hyperinsulinemic-Euglycemic Clamps. Diabetes Care 2012;35:1605-1610

English

Response to Comment on Chong Et Al. On Diabetes Reversal in NOD Mice

Science

Multidisciplinary

Philosophy of Science

History

2006

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by T. J. Pullen

Comment On: Schuit Et Al. -Cell-Specific Gene Repression: A Mechanism to Protect Against Inappropriate or Maladjusted Insulin Secretion? Diabetes 2012;61:969-975

Related publications

Response to Comment On: Schuit Et Al. -Cell-Specific Gene Repression: A Mechanism to Protect Against Inappropriate or Maladjusted Insulin Secretion? Diabetes 2012;61:969-975

Comment On: Turban Et Al. Optimal Elevation of -Cell 11 -Hydroxysteroid Dehydrogenase Type 1 Is a Compensatory Mechanism That Prevents High-Fat Diet-Induced -Cell Failure. Diabetes 2012;61:642-652

Comment on Lee Et Al. Diabetes 2015;64:2836–2846. Comment on Roberts Et Al. Diabetes 2015;64:471–484

Response to Comment On: Marquez Et Al. Low-Frequency Variants in HMGA1 Are Not Associated With Type 2 Diabetes Risk. Diabetes 2012;61:524-530

Response to Comment on Sims Et Al. Proinsulin Secretion Is a Persistent Feature of Type 1 Diabetes. Diabetes Care 2019;42:258–264

Comment On: Teeuwisse Et Al. Short-Term Caloric Restriction Normalizes Hypothalamic Neuronal Responsiveness to Glucose Ingestion in Patients With Type 2 Diabetes. Diabetes 2012;61:3255-3259

Comment on Sims Et Al. Proinsulin Secretion Is a Persistent Feature of Type 1 Diabetes. Diabetes Care 2019;42:258–264

Response to Comment On: Tam Et Al. Defining Insulin Resistance From Hyperinsulinemic-Euglycemic Clamps. Diabetes Care 2012;35:1605-1610

Response to Comment on Chong Et Al. On Diabetes Reversal in NOD Mice