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Publications by T. Schraw
Paradoxical Elevation of High-Molecular Weight Adiponectin in Acquired Extreme Insulin Resistance Due to Insulin Receptor Antibodies
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
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Exercise Training Improves Whole Body Insulin Resistance via Adiponectin Receptor 1
International Journal of Sports Medicine
Sports Medicine
Physical Therapy
Sports Science
Orthopedics
Sports Therapy
Rehabilitation
Insulin Resistance Due to Hyperglycaemia: An Adaptation Protecting Insulin-Sensitive Tissues
Diabetologia
Internal Medicine
Endocrinology
Metabolism
Diabetes
Adding Glimepiride to Current Insulin Therapy Increases High-Molecular Weight Adiponectin Levels to Improve Glycemic Control in Poorly Controlled Type 2 Diabetes
Diabetology and Metabolic Syndrome
Internal Medicine
Endocrinology
Metabolism
Diabetes
Resistin Is Associated With Biomarkers of Inflammation While Total and High-Molecular Weight Adiponectin Are Associated With Biomarkers of Inflammation, Insulin Resistance, and Endothelial Function
European Journal of Endocrinology
Medicine
Endocrinology
Metabolism
Diabetes
Metalloproteinases Damage the Insulin Receptor to Cause Insulin Resistance in Spontaneously Hypertensive Rats
Hypertension
Internal Medicine
Resistin Is Associated With Biomarkers of Inflammation While Total and High-Molecular Weight Adiponectin Are Associated With Biomarkers of Inflammation, Insulin Resistance, and Endothelial Function
European Journal of Endocrinology
Medicine
Endocrinology
Metabolism
Diabetes
Losartan Elevates the Serum High-Molecular Weight-Adiponectin Isoform and Concurrently Improves Insulin Sensitivity in Patients With Impaired Glucose Metabolism
Hypertension Research
Internal Medicine
Cardiovascular Medicine
Physiology
Cardiology
Molecular Mechanisms of Hypothalamic Insulin Resistance
International Journal of Molecular Sciences
Organic Chemistry
Molecular Biology
Theoretical Chemistry
Inorganic Chemistry
Computer Science Applications
Spectroscopy
Medicine
Catalysis
Physical
Monoclonal Antibodies to the Human Insulin Receptor That Activate Glucose Transport but Not Insulin Receptor Kinase Activity.
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
