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Publications by T.J. Welting
Expression of the Ribosomal RNA Pseudo-Uridylase Dyskerin Is Decreased by Inflammatory Mediators in Articular Chondrocytes
Osteoarthritis and Cartilage
Sports Medicine
Rheumatology
Orthopedics
Biomedical Engineering
Unraveling the Role of snoRNAs in Chondrogenic Differentiation
Osteoarthritis and Cartilage
Sports Medicine
Rheumatology
Orthopedics
Biomedical Engineering
Related publications
Cyclic Compression Loading on Three-Dimensional Tissue of Human Articular Chondrocytes Upregulates Inflammatory Mediators and Pain-Sensitizing Molecules
Osteoarthritis and Cartilage
Sports Medicine
Rheumatology
Orthopedics
Biomedical Engineering
Mouse Dyskerin Mutations Affect Accumulation of Telomerase RNA and Small Nucleolar RNA, Telomerase Activity, and Ribosomal RNA Processing
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Ectopic Expression of Cyclooxygenase-2-Induced Dedifferentiation in Articular Chondrocytes
Experimental and Molecular Medicine
Biochemistry
Medicine
Clinical Biochemistry
Molecular Biology
Molecular Medicine
Phenotypic Expression of Maternally Inherited Deafness Is Affected by RNA Modification and Cytoplasmic Ribosomal Proteins
Molecular Genetics and Metabolism
Genetics
Molecular Biology
Biochemistry
Endocrinology
Metabolism
Diabetes
Inhibition of Protein Kinase CK2 Facilitates Cellular Senescence by Inhibiting the Expression of HO-1 in Articular Chondrocytes
International Journal of Molecular Medicine
Medicine
Genetics
Transcriptome-Wide Analysis of Messenger RNA Decay in Normal and Osteoarthritic Human Articular Chondrocytes
Arthritis and Rheumatology
Rheumatology
Allergy
Immunology
Mitochondrial Changes of Articular Chondrocytes Revealed by Omics Analyses
Osteoarthritis and Cartilage
Sports Medicine
Rheumatology
Orthopedics
Biomedical Engineering
In Vitroevidence That Eukaryotic Ribosomal RNA Transcription Is Regulated by Modification of RNA Polymerase I
Nucleic Acids Research
Genetics
E2F1 and TFDP1 Regulate PITX1 Expression in Normal and Osteoarthritic Articular Chondrocytes
PLoS ONE
Multidisciplinary
