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Publications by Tabea M. Schatz
Protein Kinase C-Mediated Cardiac Troponin I S43/45 Phosphorylation Causes Contractile Dysfunction in Human Heart Failure and in Rodents
Biophysical Journal
Biophysics
Related publications
Cardiac Contractile Dysfunction and Protein Kinase C–mediated Myofilament Phosphorylation in Disease and Aging
Journal of General Physiology
Physiology
Pharmacological Inhibition of -Protein Kinase C Attenuates Cardiac Fibrosis and Dysfunction in Hypertension-Induced Heart Failure
Hypertension
Internal Medicine
Increased Phosphorylation of Cardiac Myosin Binding Protein C and Increased Expression of Troponin I in Normal Human Ageing
Biophysical Journal
Biophysics
Protein Kinase C Α and Ε Phosphorylation of Troponin and Myosin Binding Protein C Reduce Ca2+ Sensitivity in Human Myocardium
Basic Research in Cardiology
Cardiovascular Medicine
Physiology
Cardiology
020 Determination of Troponin I Phosphorylation Sites in Human Heart Muscle
Heart
Cardiovascular Medicine
Cardiology
Β-Adrenergic Augmentation of Cardiac Contractility Is Dependent on PKA-mediated Phosphorylation of Myosin-Binding Protein C and Troponin I
Journal of Physiology
Physiology
Sports Science
Cardiac Troponin I Phosphorylation at Ser149 by Protein Kinase A: A Potential Modulator of Myocardial Contractility
Biophysical Journal
Biophysics
Phosphorylation of Cardiac Troponin I by Mammalian Sterile 20-Like Kinase 1
Biochemical Journal
Biochemistry
Cell Biology
Molecular Biology
Assessment of Cardiac Effects of Protein Kinase a and Protein Kinase C Inhibitors on Myofilament-Based Contractile Activation in Failing Human Myocardium
Biophysical Journal
Biophysics