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Publications by Thomas M. Reeve
NagZ Inactivation Prevents and Reverts -Lactam Resistance, Driven by AmpD and PBP 4 Mutations, in Pseudomonas Aeruginosa
Antimicrobial Agents and Chemotherapy
Infectious Diseases
Pharmacology
Related publications
Regulation of AmpC-Driven Β-Lactam Resistance in Pseudomonas Aeruginosa: Different Pathways, Different Signaling
mSystems
Evolution
Ecology
Genetics
Molecular Biology
Biochemistry
Systematics
Microbiology
Simulation
Computer Science Applications
Behavior
Modeling
Physiology
Molecular Mechanisms of -Lactam Resistance Mediated by AmpC Hyperproduction in Pseudomonas Aeruginosa Clinical Strains
Antimicrobial Agents and Chemotherapy
Infectious Diseases
Pharmacology
Inactivation of the ampD Gene in Pseudomonas Aeruginosa Leads to Moderate-Basal-Level and Hyperinducible AmpC Beta -Lactamase Expression
Antimicrobial Agents and Chemotherapy
Infectious Diseases
Pharmacology
Proteolytic Inactivation of Cytokines by Pseudomonas Aeruginosa.
Infection and Immunity
Parasitology
Infectious Diseases
Immunology
Microbiology
Pan- -Lactam Resistance Development in Pseudomonas Aeruginosa Clinical Strains: Molecular Mechanisms, Penicillin-Binding Protein Profiles, and Binding Affinities
Antimicrobial Agents and Chemotherapy
Infectious Diseases
Pharmacology
Oligoribonuclease Contributes to Tolerance to Aminoglycoside and Β-Lactam Antibiotics by Regulating KatA in Pseudomonas Aeruginosa
Antimicrobial Agents and Chemotherapy
Infectious Diseases
Pharmacology
Synergistic Activities of Fortimicin a and Beta-Lactam Antibiotics Against Pseudomonas Aeruginosa.
Antimicrobial Agents and Chemotherapy
Infectious Diseases
Pharmacology
Resistance of Pseudomonas Aeruginosa PAO to Nalidixic Acid and Low Levels of Beta-Lactam Antibiotics: Mapping of Chromosomal Genes.
Antimicrobial Agents and Chemotherapy
Infectious Diseases
Pharmacology
An ampD Gene in Pseudomonas Aeruginosa Encodes a Negative Regulator of AmpC Β-Lactamase Expression
Antimicrobial Agents and Chemotherapy
Infectious Diseases
Pharmacology