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Publications by Toshie Nakashima
Human Wee1 Kinase Is Directly Transactivated by and Increased in Association With C-Fos/Ap-1: Rheumatoid Synovial Cells Overexpressing These Genes Go Into Aberrant Mitosis
Oncogene
Cancer Research
Genetics
Molecular Biology
Related publications
Activation of the C-Fos Enhancer by the Erk MAP Kinase Pathway Through Two Sequence Elements: The C-Fos AP-1 and P62TCF Sites
Oncogene
Cancer Research
Genetics
Molecular Biology
Biologically Active Thrombomodulin Is Synthesized by Adherent Synovial Fluid Cells and Is Elevated in Synovial Fluid of Patients With Rheumatoid Arthritis
Blood
Biochemistry
Immunology
Cell Biology
Hematology
A Mechanism of AP-1 Suppression Through Interaction of C-Fos With Lamin a/C
Genes and Development
Genetics
Developmental Biology
Increased Level of Heme Oxygenase-1 in Rheumatoid Arthritis Synovial Fluid
Modern Rheumatology
Medicine
Rheumatology
Regulation of Annexin I in Rheumatoid Synovial Cells by Glucocorticoids and Interleukin-1
Mediators of Inflammation
Immunology
Cell Biology
A Point Mutation at Tyrosine-809 in the Human Colony-Stimulating Factor 1 Receptor Impairs Mitogenesis Without Abrogating Tyrosine Kinase Activity, Association With Phosphatidylinositol 3-Kinase, or Induction of C-Fos and junB Genes.
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Expression of Interleukin-1 and Interleukin-1 Receptor Antagonist by Human Rheumatoid Synovial Tissue Macrophages
Clinical Immunology and Immunopathology
Increased Expression of miR-551a by C-Fos Reduces Focal Adhesion Kinase (FAK) Levels and Blocks Tumorigenesis
Molecular and Cellular Biology
Cell Biology
Molecular Biology
Interleukin-34 Produced by Human Fibroblast-Like Synovial Cells in Rheumatoid Arthritis Supports Osteoclastogenesis
Arthritis Research and Therapy
Rheumatology
Allergy
Immunology
