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Publications by U Resch

Α-Catulin, a Rho Signalling Component, Can Regulate NF-κB Through Binding to IKK-β, and Confers Resistance to Apoptosis

Oncogene

English

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IKK/NF-κB Signaling Contributes to Glioblastoma Stem Cell Maintenance

Oncotarget

Oncology

2016

English

Bile Acids Regulate RANTES Gene Expression Through Its Cognate NF-κB Binding Sites

Biochemical and Biophysical Research Communications

English

Suppression of NF-κB and NF-κB-Regulated Gene Expression by Apigenin Through IκBα and IKK Pathway in TRAMP Mice

PLoS ONE

Multidisciplinary

2015

English

Inactivation of BAD by IKK Inhibits TNFα-Induced Apoptosis Independently of NF-κB Activation

English

Lipopolysaccharide Induces Rac1-Dependent Reactive Oxygen Species Formation and Coordinates Tumor Necrosis Factor-Α Secretion Through IKK Regulation of NF-κB

Journal of Biological Chemistry

English

Genistein Sensitizes Bladder Cancer Cells to HCPT Treatment in Vitro and in Vivo via ATM/NF-κB/IKK Pathway-Induced Apoptosis

PLoS ONE

Multidisciplinary

2013

English

Biomechanical Signals Inhibit IKK Activity to Attenuate NF-κB Transcription Activity in Inflamed Chondrocytes

Arthritis & Rheumatism

2007

English

Biomechanical Thresholds Regulate Inflammation Through the NF-κB Pathway: Experiments and Modeling

PLoS ONE

Multidisciplinary

2009

English

Exposure to TNF‑α Combined With TGF‑β Induces Carcinogenesis in Vitro via NF-κB/Twist Axis

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by U Resch

Α-Catulin, a Rho Signalling Component, Can Regulate NF-κB Through Binding to IKK-β, and Confers Resistance to Apoptosis

Related publications

IKK/NF-κB Signaling Contributes to Glioblastoma Stem Cell Maintenance

Bile Acids Regulate RANTES Gene Expression Through Its Cognate NF-κB Binding Sites

Suppression of NF-κB and NF-κB-Regulated Gene Expression by Apigenin Through IκBα and IKK Pathway in TRAMP Mice

Inactivation of BAD by IKK Inhibits TNFα-Induced Apoptosis Independently of NF-κB Activation

Lipopolysaccharide Induces Rac1-Dependent Reactive Oxygen Species Formation and Coordinates Tumor Necrosis Factor-Α Secretion Through IKK Regulation of NF-κB

Genistein Sensitizes Bladder Cancer Cells to HCPT Treatment in Vitro and in Vivo via ATM/NF-κB/IKK Pathway-Induced Apoptosis

Biomechanical Signals Inhibit IKK Activity to Attenuate NF-κB Transcription Activity in Inflamed Chondrocytes

Biomechanical Thresholds Regulate Inflammation Through the NF-κB Pathway: Experiments and Modeling

Exposure to TNF‑α Combined With TGF‑β Induces Carcinogenesis in Vitro via NF-κB/Twist Axis