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Publications by V Walf-Vorderwülbecke
The AAA+ ATPase RUVBL2 Is a Critical Mediator of MLL-AF9 Oncogenesis
Leukemia
Cancer Research
Oncology
Anesthesiology
Pain Medicine
Hematology
Related publications
G1 Cell-Cycle Arrest and Apoptosis by Histone Deacetylase Inhibition in MLL-AF9 Acute Myeloid Leukemia Cells Is P21 Dependent and MLL-AF9 Independent
Leukemia
Cancer Research
Oncology
Anesthesiology
Pain Medicine
Hematology
Mouse Af9 Is a Controller of Embryo Patterning, Like Mll, Whose Human Homologue Fuses With AF9 After Chromosomal Translocation in Leukemia
Molecular and Cellular Biology
Cell Biology
Molecular Biology
The AAA+ ATPase P97, a Cellular Multitool
Biochemical Journal
Biochemistry
Cell Biology
Molecular Biology
Cilia-Associated Cellular Function of the AAA ATPases RUVBL1 and RUVBL2
Molecular and Cellular Pediatrics
Child Health
Pediatrics
Perinatology
Molecular Biology
Cell Biology
Structure of the MLL CXXC Domain–DNA Complex and Its Functional Role in MLL-AF9 Leukemia
Nature Structural and Molecular Biology
Structural Biology
Molecular Biology
PECAM-1 Is a Critical Mediator of Atherosclerosis
DMM Disease Models and Mechanisms
Immunology
Molecular Biology
Biochemistry
Microbiology
Neuroscience
Medicine
Genetics
Hoxa9 Influences the Phenotype but Not the Incidence of MLL-Af9 Fusion Gene Leukemia
Blood
Biochemistry
Immunology
Cell Biology
Hematology
SKD1 AAA ATPase-Dependent Endosomal Transport Is Involved in Autolysosome Formation
Cell Structure and Function
Medicine
Cell Biology
Molecular Biology
Physiology
FAK Is a Critical Mediator of GNAQ-Driven Uveal Melanoma
Cancer Discovery
Oncology
