Amanote Research
Register
Sign In
Discover open access scientific publications
Search, annotate, share and cite publications
Publications by V. Braun
Phosphatidylinositol 3-Kinase Inhibition Broadly Sensitizes Glioblastoma Cells to Death Receptor- And Drug-Induced Apoptosis
Cancer Research
Cancer Research
Oncology
TonB-dependent Maltose Transport by Caulobacter Crescentus
Microbiology
Microbiology
Related publications
Inhibition of Phosphatidylinositol 3-Kinase by PX-866 Suppresses Temozolomide-Induced Autophagy and Promotes Apoptosis in Glioblastoma Cells
Molecular Medicine
Molecular Medicine
Genetics
Molecular Biology
Inhibition of Casein Kinase-2 Induces P53-Dependent Cell Cycle Arrest and Sensitizes Glioblastoma Cells to Tumor Necrosis Factor (TNFα)-induced Apoptosis Through SIRT1 Inhibition
Cell Death and Disease
Molecular Neuroscience
Immunology
Cell Biology
Cancer Research
Cellular
Medicine
Selective Inhibition of Histone Deacetylases Sensitizes Malignant Cells to Death Receptor Ligands
Molecular Cancer Therapeutics
Cancer Research
Oncology
Inhibition of MEK/ERK1/2 Sensitizes Lymphoma Cells to Sorafenib-Induced Apoptosis
Leukemia Research
Cancer Research
Oncology
Hematology
PARP Inhibition Sensitizes P53-Deficient Breast Cancer Cells to Doxorubicin-Induced Apoptosis
Biochemical Journal
Biochemistry
Cell Biology
Molecular Biology
Inhibition of Phosphatidylinositol 3-Kinase Causes Apoptosis in Retinoic Acid-Differentiated HL-60 Leukemia Cells
Cell Cycle
Medicine
Developmental Biology
Cell Biology
Molecular Biology
Tyrosine Kinase Eph Receptor A6 Sensitizes Glioma‐initiating Cells Towards Bone Morphogenetic Protein‐induced Apoptosis
Cancer Science
Cancer Research
Medicine
Oncology
Luteolin Sensitizes Human Liver Cancer Cells to TRAIL‑induced Apoptosis via Autophagy and JNK‑mediated Death Receptor 5 Upregulation
International Journal of Oncology
Cancer Research
Oncology
Inhibition of Autophagy Triggers Melatonin-Induced Apoptosis in Glioblastoma Cells
BMC Neuroscience
Neuroscience
Cellular
Molecular Neuroscience
