Amanote Research
Register
Sign In
Discover open access scientific publications
Search, annotate, share and cite publications
Publications by Vasileios Armenis
ΙκΒ Kinase Α Is Required for Development and Progression of KRAS-mutant Lung Adenocarcinoma.
Cancer Research
Cancer Research
Oncology
Related publications
Coxsackievirus Type B3 Is a Potent Oncolytic Virus Against KRAS-Mutant Lung Adenocarcinoma
Molecular Therapy - Oncolytics
Cancer Research
Oncology
Molecular Medicine
Pharmacology
Immunohistochemical Loss of LKB1 Is a Biomarker for More Aggressive Biology in KRAS-Mutant Lung Adenocarcinoma
Clinical Cancer Research
Cancer Research
Oncology
Inhibitor of Differentiation-1 (Id1) Sustains Mutant KRAS-driven Progression, Maintenance, and Metastasis of Lung Adenocarcinoma via Regulation of a FOSL1 Network
Cancer Research
Cancer Research
Oncology
Coordinate Direct Input of Both KRAS and IGF1 Receptor to Activation of PI3 Kinase in KRAS -Mutant Lung Cancer
Cancer Discovery
Oncology
P2.01-09 Targetable Genomic Alterations in KRAS Mutant Lung Adenocarcinoma by Targeted Next Generation Sequencing
Journal of Thoracic Oncology
Medicine
Oncology
Respiratory Medicine
Pulmonary
Growth Inhibition of KRAS‑�and EGFR‑mutant Lung Adenocarcinoma by Cosuppression of STAT3 and the SRC/ARHGAP35 Axis
Oncology Reports
Medicine
Cancer Research
Oncology
Targeting Pathways Downstream of KRAS in Lung Adenocarcinoma
Pharmacogenomics
Molecular Medicine
Genetics
Pharmacology
ASAP3 Is a Downstream Target of HIF-1α and Is Critical for Progression of Lung Adenocarcinoma
OncoTargets and Therapy
Oncology
Pharmacology
Targeting Oncogenic Protein Kinase Cι for Treatment of Mutant KRAS LADC
Small GTPases
Biochemistry
Cell Biology
