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Publications by William N Pappano
Genetic Inhibition of the Atypical Kinase Wee1 Selectively Drives Apoptosis of P53 Inactive Tumor Cells
BMC Cancer
Cancer Research
Oncology
Genetics
Related publications
Small-Molecule Inhibition of Wee1 Kinase by MK-1775 Selectively Sensitizes P53-Deficient Tumor Cells to DNA-damaging Agents
Molecular Cancer Therapeutics
Cancer Research
Oncology
Multiple Defects Sensitize P53-Deficient Head and Neck Cancer Cells to the WEE1 Kinase Inhibition
Molecular Cancer Research
Cancer Research
Oncology
Molecular Biology
Inhibition of Casein Kinase-2 Induces P53-Dependent Cell Cycle Arrest and Sensitizes Glioblastoma Cells to Tumor Necrosis Factor (TNFα)-induced Apoptosis Through SIRT1 Inhibition
Cell Death and Disease
Molecular Neuroscience
Immunology
Cell Biology
Cancer Research
Cellular
Medicine
Inhibition of C-Jun N-Terminal Kinase 2 Expression Suppresses Growth and Induces Apoptosis of Human Tumor Cells in a P53-Dependent Manner
Molecular and Cellular Biology
Cell Biology
Molecular Biology
Flavopiridol Induces P53 via Initial Inhibition of Mdm2 and P21 And, Independently of P53, Sensitizes Apoptosis-Reluctant Cells to Tumor Necrosis Factor
Cancer Research
Cancer Research
Oncology
WEE1 Kinase Inhibition Enhances the Radiation Response of Diffuse Intrinsic Pontine Gliomas
Molecular Cancer Therapeutics
Cancer Research
Oncology
E2f-1 Cooperates With Topoisomerase II Inhibition and DNA Damage to Selectively Augment P53-Independent Apoptosis.
Molecular and Cellular Biology
Cell Biology
Molecular Biology
Ubiquitous Induction of P53 in Tumor Cells by Antisense Inhibition of MDM2 Expression
Molecular Medicine
Molecular Medicine
Genetics
Molecular Biology
RITA (Reactivating P53 and Inducing Tumor Apoptosis) Is Efficient Against TP53 Abnormal Myeloma Cells Independently of the P53 Pathway
BMC Cancer
Cancer Research
Oncology
Genetics
