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Publications by Woldeab B Haile
The Interaction Between Tumor Necrosis Factor-Like Weak Inducer of Apoptosis and Its Receptor Fibroblast Growth Factor-Inducible 14 Promotes the Recruitment of Neutrophils Into the Ischemic Brain
Journal of Cerebral Blood Flow and Metabolism
Neurology
Cardiovascular Medicine
Cardiology
Related publications
The Relationship Between Soluble Tumor Necrosis Factor-Like Weak Inducer of Apoptosis Levels and Cardiac Functions in Peritoneal Dialysis Patients
European Journal of Inflammation
Allergy
Immunology
Cryptotanshinone Attenuates Airway Remodeling by Inhibiting Crosstalk Between Tumor Necrosis Factor-Like Weak Inducer of Apoptosis and Transforming Growth Factor Beta 1 Signaling Pathways in Asthma
Frontiers in Pharmacology
Pharmacology
Tumor Necrosis Factor-Like Weak Inducer of Apoptosis or Fn14 Deficiency Reduce Elastase Perfusion-Induced Aortic Abdominal Aneurysm in Mice
Journal of the American Heart Association
Cardiovascular Medicine
Cardiology
Tumor Necrosis Factor-Like Weak Inducer of Apoptosis (TWEAK) Promotes Glioma Cell Invasion Through Induction of NF-κB-inducing Kinase (NIK) and Noncanonical NF-κB Signaling
Molecular Cancer
Cancer Research
Oncology
Molecular Medicine
Interaction Between the mRNA of the 55-kDa Tumor Necrosis Factor Receptor and Cellular Proteins
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Investigating the Interaction Between Osteoprotegerin and Receptor Activator of NF-κB or Tumor Necrosis Factor-Related Apoptosis-Inducing Ligand
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Identification of a Novel Activation-Inducible Protein of the Tumor Necrosis Factor Receptor Superfamily and Its Ligand
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Aldosterone Abrogates Nuclear Factor B-Mediated Tumor Necrosis Factor Production in Human Neutrophils via the Mineralocorticoid Receptor
Hypertension
Internal Medicine
The Tumor Necrosis Factor Receptor 2 Signal Transducers TRAF2 and C-Iap1 Are Components of the Tumor Necrosis Factor Receptor 1 Signaling Complex
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
