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Publications by Xueshi Wang

Astragalin-Induced Cell Death Is Caspase-Dependent and Enhances the Susceptibility of Lung Cancer Cells to Tumor Necrosis Factor by Inhibiting the NF-кB Pathway

Oncotarget

Oncology

2017

English

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Tumor Necrosis Factor-Induced Apoptosis Is Mediated by a CrmA-sensitive Cell Death Pathway.

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

1995

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Lithium Sensitizes Tumor Cells in an NF-κB-independent Way to Caspase Activation and Apoptosis Induced by Tumor Necrosis Factor (TNF)

Journal of Biological Chemistry

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Zoledronate Sensitizes Endothelial Cells to Tumor Necrosis Factor-Induced Programmed Cell Death

Journal of Biological Chemistry

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Resistance to Tumor Necrosis Factor-Induced Cell Death Mediated by PMCA4 Deficiency

Molecular and Cellular Biology

Cell Biology

Molecular Biology

2001

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Caspase-10 Sensitizes Breast Carcinoma Cells to TRAIL-Induced but Not Tumor Necrosis Factor-Induced Apoptosis in a Caspase- 3-Dependent Manner

Molecular and Cellular Biology

Cell Biology

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2005

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Endothelial Cell Death Induced by Tumor Necrosis Factor-Α Is Inhibited by the BCL-2 Family Member, A1

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Cordyceps Militaris Induces Tumor Cell Death via the Caspase-Dependent Mitochondrial Pathway in HepG2 and McF-7 Cells

Molecular Medicine Reports

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TSA-induced Cell Death in Prostate Cancer Cell Lines Is Caspase-2 Dependent and Involves the PIDDosome

Cancer Biology and Therapy

English

Embelin Suppresses Osteoclastogenesis Induced by Receptor Activator of NF- B Ligand and Tumor Cells in Vitro Through Inhibition of the NF- B Cell Signaling Pathway

Molecular Cancer Research

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Xueshi Wang

Astragalin-Induced Cell Death Is Caspase-Dependent and Enhances the Susceptibility of Lung Cancer Cells to Tumor Necrosis Factor by Inhibiting the NF-&#x043A;B Pathway

Related publications

Tumor Necrosis Factor-Induced Apoptosis Is Mediated by a CrmA-sensitive Cell Death Pathway.

Lithium Sensitizes Tumor Cells in an NF-κB-independent Way to Caspase Activation and Apoptosis Induced by Tumor Necrosis Factor (TNF)

Zoledronate Sensitizes Endothelial Cells to Tumor Necrosis Factor-Induced Programmed Cell Death

Resistance to Tumor Necrosis Factor-Induced Cell Death Mediated by PMCA4 Deficiency

Caspase-10 Sensitizes Breast Carcinoma Cells to TRAIL-Induced but Not Tumor Necrosis Factor-Induced Apoptosis in a Caspase- 3-Dependent Manner

Endothelial Cell Death Induced by Tumor Necrosis Factor-Α Is Inhibited by the BCL-2 Family Member, A1

Cordyceps Militaris Induces Tumor Cell Death via the Caspase-Dependent Mitochondrial Pathway in HepG2 and McF-7 Cells

TSA-induced Cell Death in Prostate Cancer Cell Lines Is Caspase-2 Dependent and Involves the PIDDosome

Embelin Suppresses Osteoclastogenesis Induced by Receptor Activator of NF- B Ligand and Tumor Cells in Vitro Through Inhibition of the NF- B Cell Signaling Pathway

Astragalin-Induced Cell Death Is Caspase-Dependent and Enhances the Susceptibility of Lung Cancer Cells to Tumor Necrosis Factor by Inhibiting the NF-кB Pathway