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Publications by Y. Ebina
Platelet-Derived Growth Factor Triggers Translocation of the Insulin-Regulatable Glucose Transporter (Type 4) Predominantly Through Phosphatidylinositol 3-Kinase Binding Sites on the Receptor.
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Resistance of Human Cells to Tumorigenesis Induced by Cloned Transforming Genes.
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
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Interaction of Phosphatidylinositol 3-Kinase-Associated P85 With Epidermal Growth Factor and Platelet-Derived Growth Factor Receptors.
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Antiapoptotic Signalling by the Insulin-Like Growth Factor I Receptor, Phosphatidylinositol 3-Kinase, and Akt.
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The C-Terminal SH2 Domain of P85 Accounts for the High Affinity and Specificity of the Binding of Phosphatidylinositol 3-Kinase to Phosphorylated Platelet-Derived Growth Factor Beta Receptor.
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Tyrosine Mutations Within the Alpha Platelet-Derived Growth Factor Receptor Kinase Insert Domain Abrogate Receptor-Associated Phosphatidylinositol-3 Kinase Activity Without Affecting Mitogenic or Chemotactic Signal Transduction.
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Type I Transforming Growth Factor Β Receptor Binds to and Activates Phosphatidylinositol 3-Kinase
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Platelet-Derived Growth Factor Activates Protein Kinase C Epsilon Through Redundant and Independent Signaling Pathways Involving Phospholipase C Gamma or Phosphatidylinositol 3-Kinase.
Proceedings of the National Academy of Sciences of the United States of America
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Platelet Derived Growth Factor a (PDGFA; PDGF1); Platelet Derived Growth Factor Receptor (PDGFR)
Science-Business eXchange
E5 Oncoprotein Mutants Activate Phosphoinositide 3-Kinase Independently of Platelet-Derived Growth Factor Receptor Activation
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Requirement for Phosphatidylinositol 3′-Kinase Activity in Platelet-Derived Growth Factor-Stimulated Tyrosine Phosphorylation of P125 Focal Adhesion Kinase and Paxillin
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