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Publications by Yasumitsu TAKADA

Changes in Receptor Binding, Biological Activity and Immunoreactivity of Insulin Caused by Replacing the Residues B23-B26 Vvith Alanine

Biomedical Research

English

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Effect of Replacing Glutamic Residues Upon the Biological Activity and Stability of the Circular Enterocin AS-48

Journal of Antimicrobial Chemotherapy

English

Alanine-Scanning Mutagenesis of Plasmatocyte Spreading Peptide Identifies Critical Residues for Biological Activity

Journal of Biological Chemistry

English

Alanine-Scanning Mutagenesis Reveals Residues Involved in Binding of Pap-3-Encoded Pili.

Journal of Bacteriology

Microbiology

Molecular Biology

1994

English

Regulation of Upstream Binding Factor 1 Activity by Insulin-Like Growth Factor I Receptor Signaling

Journal of Biological Chemistry

English

Azapbilones With Endothelin Receptor Binding Activity Produced by Penicillium Sclerotiorum: Taxonomy, Fermentation, Isolation, Structure Elucidation and Biological Activity.

Journal of Antibiotics

Drug Discovery

Pharmacology

1995

English

Channel Gating of the Glycine Receptor Changes Accessibility to Residues Implicated in Receptor Potentiation by Alcohols and Anesthetics

Journal of Biological Chemistry

English

Insulin Receptor Is an Insulin-Dependent Tyrosine Protein Kinase: Copurification of Insulin-Binding Activity and Protein Kinase Activity to Homogeneity From Human Placenta.

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

1984

English

Anion-Coordinating Residues at Binding Site 1 Are Essential for the Biological Activity of the Diphtheria Toxin Repressor

Infection and Immunity

English

Functional Reconstitution of Insulin Receptor Binding Site From Non-Binding Receptor Fragments

Journal of Biological Chemistry

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Yasumitsu TAKADA

Changes in Receptor Binding, Biological Activity and Immunoreactivity of Insulin Caused by Replacing the Residues B23-B26 Vvith Alanine

Related publications

Effect of Replacing Glutamic Residues Upon the Biological Activity and Stability of the Circular Enterocin AS-48

Alanine-Scanning Mutagenesis of Plasmatocyte Spreading Peptide Identifies Critical Residues for Biological Activity

Alanine-Scanning Mutagenesis Reveals Residues Involved in Binding of Pap-3-Encoded Pili.

Regulation of Upstream Binding Factor 1 Activity by Insulin-Like Growth Factor I Receptor Signaling

Azapbilones With Endothelin Receptor Binding Activity Produced by Penicillium Sclerotiorum: Taxonomy, Fermentation, Isolation, Structure Elucidation and Biological Activity.

Channel Gating of the Glycine Receptor Changes Accessibility to Residues Implicated in Receptor Potentiation by Alcohols and Anesthetics

Insulin Receptor Is an Insulin-Dependent Tyrosine Protein Kinase: Copurification of Insulin-Binding Activity and Protein Kinase Activity to Homogeneity From Human Placenta.

Anion-Coordinating Residues at Binding Site 1 Are Essential for the Biological Activity of the Diphtheria Toxin Repressor

Functional Reconstitution of Insulin Receptor Binding Site From Non-Binding Receptor Fragments