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Publications by Youji MITSUI

P53-Independent Upregulation of P21WAF1 in NIH 3T3 Cells Malignantly Transformed by Mot-2

Cell Research

Cell Biology

Molecular Biology

2001

English

Identification and Characterization of Molecular Interactions Between Glucose-Regulated Proteins (GRPs) Mortalin/Grp75/Peptide-Binding Protein 74 (PBP74) and GRP94

English

Related publications

Growth Factor Requirements of Oncogene-Transformed NIH 3T3 and BALB/c 3T3 Cells Cultured in Defined Media.

Molecular and Cellular Biology

Cell Biology

Molecular Biology

1986

English

Induction of Apoptosis by Selected Natural Products in V-CBL Transformed COS and NIH 3T3 Cells

International Journal of Cancer Research

Cancer Research

Oncology

2005

English

Store Operated Calcium Entry in NIH-3T3 Cells

Journal of Medical Investigation

English

HDAC4 Represses P21WAF1/Cip1 Expression in Human Cancer Cells Through a Sp1-Dependent, P53-Independent Mechanism

English

Nimustine Induces DNA Breaks and Crosslinks in NIH/3T3 Cells

International Journal of Bioscience, Biochemistry and Bioinformatics

2013

English

Compartmentalized Ras Proteins Transform NIH 3T3 Cells With Different Efficiencies

Molecular and Cellular Biology

Cell Biology

Molecular Biology

2010

English

Pleiotrophin Transforms NIH 3T3 Cells and Induces Tumors in Nude Mice.

Proceedings of the National Academy of Sciences of the United States of America

Multidisciplinary

1993

English

Inactivation of Tumor Suppressor P53 by Mot-2, a Hsp70 Family Member

Journal of Biological Chemistry

English

Dorrigocins: Novel Antifungal Antibiotics That Change the Morphology of Ras-Transformed NIH/3T3 Cells to That of Normal Cells. II. Isolation and Elucidation of Structures.

Journal of Antibiotics

Drug Discovery

Pharmacology

1994

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Youji MITSUI

P53-Independent Upregulation of P21WAF1 in NIH 3T3 Cells Malignantly Transformed by Mot-2

Identification and Characterization of Molecular Interactions Between Glucose-Regulated Proteins (GRPs) Mortalin/Grp75/Peptide-Binding Protein 74 (PBP74) and GRP94

Related publications

Growth Factor Requirements of Oncogene-Transformed NIH 3T3 and BALB/c 3T3 Cells Cultured in Defined Media.

Induction of Apoptosis by Selected Natural Products in V-CBL Transformed COS and NIH 3T3 Cells

Store Operated Calcium Entry in NIH-3T3 Cells

HDAC4 Represses P21WAF1/Cip1 Expression in Human Cancer Cells Through a Sp1-Dependent, P53-Independent Mechanism

Nimustine Induces DNA Breaks and Crosslinks in NIH/3T3 Cells

Compartmentalized Ras Proteins Transform NIH 3T3 Cells With Different Efficiencies

Pleiotrophin Transforms NIH 3T3 Cells and Induces Tumors in Nude Mice.

Inactivation of Tumor Suppressor P53 by Mot-2, a Hsp70 Family Member

Dorrigocins: Novel Antifungal Antibiotics That Change the Morphology of Ras-Transformed NIH/3T3 Cells to That of Normal Cells. II. Isolation and Elucidation of Structures.