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Publications in Internal Medicine

Ghrelin, an Endogenous Growth Hormone Secretagogue, Is a Novel Orexigenic Peptide That Antagonizes Leptin Action Through the Activation of Hypothalamic Neuropeptide Y/Y1 Receptor Pathway

Diabetes

English

Response to Comment On: Chang Et Al. (2007) Association Study of the Genetic Polymorphisms of the Transcription Factor 7-Like 2 (TCF7L2) Gene and Type 2 Diabetes in the Chinese Population: Diabetes 56:2631 2637

English

Public Health Genomics Approach to Type 2 Diabetes

English

Polymorphism Screening of Four Genes Encoding Advanced Glycation End-Product Putative Receptors: Association Study With Nephropathy in Type 1 Diabetic Patients

English

Interleukin-1beta-Induced Alteration in a Beta-Cell Phenotype Can Reduce Cellular Sensitivity to Conditions That Cause Necrosis but Not to Cytokine-Induced Apoptosis

English

Response to Comment On: Thallas-Bonke Et Al. (2008) Inhibition of NADPH Oxidase Prevents Advanced Glycation End Product-Mediated Damage in Diabetic Nephropathy Through a Protein Kinase C- -Dependent Pathway: Diabetes 57:460-469, 2008

English

Diabetic Glomerulosclerosis

English

Changes in Regional Brain 18f-Fluorodeoxyglucose Uptake at Hypoglycemia in Type 1 Diabetic Men Associated With Hypoglycemia Unawareness and Counter-Regulatory Failure

English

Erratum

English

The Pathogenesis of Staphylococcus Aureus Infection in the Diabetic NOD Mouse

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications in Internal Medicine

Ghrelin, an Endogenous Growth Hormone Secretagogue, Is a Novel Orexigenic Peptide That Antagonizes Leptin Action Through the Activation of Hypothalamic Neuropeptide Y/Y1 Receptor Pathway

Response to Comment On: Chang Et Al. (2007) Association Study of the Genetic Polymorphisms of the Transcription Factor 7-Like 2 (TCF7L2) Gene and Type 2 Diabetes in the Chinese Population: Diabetes 56:2631 2637

Public Health Genomics Approach to Type 2 Diabetes

Polymorphism Screening of Four Genes Encoding Advanced Glycation End-Product Putative Receptors: Association Study With Nephropathy in Type 1 Diabetic Patients

Interleukin-1beta-Induced Alteration in a Beta-Cell Phenotype Can Reduce Cellular Sensitivity to Conditions That Cause Necrosis but Not to Cytokine-Induced Apoptosis

Response to Comment On: Thallas-Bonke Et Al. (2008) Inhibition of NADPH Oxidase Prevents Advanced Glycation End Product-Mediated Damage in Diabetic Nephropathy Through a Protein Kinase C- -Dependent Pathway: Diabetes 57:460-469, 2008

Diabetic Glomerulosclerosis

Changes in Regional Brain 18f-Fluorodeoxyglucose Uptake at Hypoglycemia in Type 1 Diabetic Men Associated With Hypoglycemia Unawareness and Counter-Regulatory Failure

Erratum

The Pathogenesis of Staphylococcus Aureus Infection in the Diabetic NOD Mouse