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Publications in Internal Medicine
Effects of Sex and Hormone Replacement Therapy Use on the Prevalence of Isolated Impaired Fasting Glucose and Isolated Impaired Glucose Tolerance in Subjects With a Family History of Type 2 Diabetes
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
Resistin Impairs Insulin-Evoked Vasodilation
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
Human Small Intestine Facilitative Fructose/Glucose Transporter (GLUT5) Is Also Present in Insulin-Responsive Tissues and Brain: Investigation of Biochemical Characteristics and Translocation
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
Glucokinase Is an Integral Component of the Insulin Granules in Glucose-Responsive Insulin Secretory Cells and Does Not Translocate During Glucose Stimulation
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
The Novel Imidazoline Compound BL11282 Potentiates Glucose-Induced Insulin Secretion in Pancreatic -Cells in the Absence of Modulation of KATP Channel Activity
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
Tumor Necrosis Factor- Induces Skeletal Muscle Insulin Resistance in Healthy Human Subjects via Inhibition of Akt Substrate 160 Phosphorylation
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
Gamma Globulin Levels Predict Type 2 Diabetes in the Pima Indian Population
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
Increasing Dietary Leucine Intake Reduces Diet-Induced Obesity and Improves Glucose and Cholesterol Metabolism in Mice via Multimechanisms
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
Phosphatidylinositol 3-Kinase-Dependent Activation of Akt, an Essential Signal for Hyperthermia-Induced Heat-Shock Protein 72, Is Attenuated in Streptozotocin-Induced Diabetic Heart
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
Aldose Reductase-Deficient Mice Are Protected From Delayed Motor Nerve Conduction Velocity, Increased C-Jun NH2-Terminal Kinase Activation, Depletion of Reduced Glutathione, Increased Superoxide Accumulation, and DNA Damage
Diabetes
Internal Medicine
Endocrinology
Metabolism
Diabetes
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