Amanote Research
Register
Sign In
Discover open access scientific publications
Search, annotate, share and cite publications
The Short Isoform of PML-RARα Activates the NRF2/HO-1 Pathway Through a Direct Interaction With NRF2
FEBS Letters
Genetics
Cell Biology
Molecular Biology
Biochemistry
Structural Biology
Biophysics
Human Immunodeficiency Virus Type 1 Coreceptor Preferences Determine Target T-Cell Depletion and Cellular Tropism in Human Lymphoid Tissue
Journal of Virology
Insect Science
Immunology
Microbiology
Virology
Elderly Non-Hodgkin's Lymphoma Presenting With Pulmonary Squamous Cell Carcinoma as a Complication of Chemotherapy for Malignant Lymphoma.
Japanese Journal of Geriatrics
Gerontology
Geriatrics
Mechanical Stimulation of Polycystin-1 Induces Human Osteoblastic Gene Expression via Potentiation of the Calcineurin/Nfat Signaling Axis
Cellular and Molecular Life Sciences
Molecular Neuroscience
Molecular Medicine
Cell Biology
Molecular Biology
Pharmacology
Cellular
The Role of Scaffold Proteins in MEK/ERK Signalling: Figure 1
Biochemical Society Transactions
Biochemistry
Metabotropic Glutamate Receptor 1-Induced Upregulation of NMDA Receptor Current: Mediation Through the Pyk2/SRC-Family Kinase Pathway in Cortical Neurons
Journal of Neuroscience
Neuroscience
Cyclic Nucleotide Signalling Compartmentation by Phosphodiesterases in Cultured Vascular Smooth Muscle Cells
British Journal of Pharmacology
Pharmacology
Combined Treatment With Sinomenine and Acupuncture on Collagen‑induced Arthritis Through the NF‑κB and MAPK Signaling Pathway
Oncology Letters
Cancer Research
Oncology
A Human Recombinant Fab Identifies a Human Immunodeficiency Virus Type 1-Induced Conformational Change in Cell Surface-Expressed CD4.
Journal of Virology
Insect Science
Immunology
Microbiology
Virology
Carbon Tetrachloride-Induced Hepatic Injury Through Formation of Oxidized Diacylglycerol and Activation of the PKC/NF-κB Pathway
Laboratory Investigation
Forensic Medicine
Pathology
Cell Biology
Molecular Biology
‹
766
767
768
769
770
771
772
›