Reductions in Amyloid-Β-Derived Neuroinflammation, With Minocycline, Restore Cognition but Do Not Significantly Affect Tau Hyperphosphorylation

Journal of Alzheimer's Disease - Netherlands
doi 10.3233/jad-2010-100204
Full Text
Abstract

Available in full text

Date
Authors
Publisher

IOS Press

Related search

The Spleen Tyrosine Kinase (Syk) Regulates Alzheimer Amyloid-Β Production and Tau Hyperphosphorylation

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
2014English

Amyloid-Β and Tau

JAMA Neurology
Neurology
2014English

Region-Specific Vulnerability of Neuroinflammation, Oxidative Stress and Tau Hyperphosphorylation in Experimental Diabetes

Juntendo Medical Journal
2016English

Neurons Derived From Sporadic Alzheimer’s Disease iPSCs Reveal Elevated TAU Hyperphosphorylation, Increased Amyloid Levels, and GSK3B Activation

Alzheimer's Research and Therapy
NeurologyCognitive Neuroscience
2017English

Succinylation Links Metabolic Reductions to Amyloid and Tau Pathology

2019English

Hyperphosphorylation-Induced Tau Oligomers

Frontiers in Neurology
Neurology
2013English

Loss of Hsp110 Leads to Age-Dependent Tau Hyperphosphorylation and Early Accumulation of Insoluble Amyloid

Molecular and Cellular Biology
Cell BiologyMolecular Biology
2010English

Brain Morphology, Cognition, and Β-Amyloid in Older Adults With Superior Memory Performance

Neurobiology of Aging
AgingGerontologyDevelopmental BiologyGeriatricsNeuroscienceNeurology
2018English

Tau Passive Immunization Inhibits Not Only Tau but Also Aβ Pathology

Alzheimer's Research and Therapy
NeurologyCognitive Neuroscience
2017English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy