A Neurotoxic and Gliotrophic Fragment of the Prion Protein Increases Plasma Membrane Microviscosity

Neurobiology of Disease - United States
doi 10.1006/nbdi.1997.0133
Full Text
Abstract

Available in full text

Categories
Neurology
Date
Authors
Publisher

Elsevier BV

Related search

Sphingosine Kinase-Dependent Migration of Immature Dendritic Cells in Response to Neurotoxic Prion Protein Fragment

Journal of Virology
Insect ScienceImmunologyMicrobiologyVirology
2003English

HIV-1 Tat Protein Increases Microglial Outward K+ Current and Resultant Neurotoxic Activity

PLoS ONE
Multidisciplinary
2013English

Prion Infections and Anti-PrP Antibodies Trigger Converging Neurotoxic Pathways

PLoS Pathogens
ImmunologyMolecular BiologyVirologyMicrobiologyParasitologyGenetics
2015English

Elevated cAMP Increases Aquaporin-3 Plasma Membrane Diffusion

American Journal of Physiology - Cell Physiology
PhysiologyCell Biology
2014English

Metal-Binding Ability of Human Prion Protein Fragment Peptides Analyzed by Column Switch HPLC

Chemical and Pharmaceutical Bulletin
MedicineDrug DiscoveryChemistry
2011English

PMP1 18-38, a Yeast Plasma Membrane Protein Fragment, Binds Phosphatidylserine From Bilayer Mixtures With Phosphatidylcholine: A 2h-NMR Study

Biophysical Journal
Biophysics
2000English

Most Pathogenic Mutations Do Not Alter the Membrane Topology of the Prion Protein

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
2000English

Erythrocyte Membrane Microviscosity and Phospholipid Composition in Lead Workers.

Occupational and Environmental Medicine
EnvironmentalPublic HealthOccupational Health
1987English

Membrane-Potential-Dependent Changes of the Lipid Microviscosity of Mitochondria and Phospholipid Vesicles

Biochemical Journal
BiochemistryCell BiologyMolecular Biology
1984English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy