(PDF) Type-2 Diabetic Leprdb/Db Mice Show a Defective

Type-2 Diabetic Leprdb/Db Mice Show a Defective Microvascular Phenotype Under Basal Conditions and an Impaired Response to Angiogenesis Gene Therapy in the Setting of Limb Ischemia

Frontiers in Bioscience - Landmark - United States

doi 10.2741/2205

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Date

January 1, 2007

Authors

Costanza Emanueli

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Frontiers in Bioscience

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Type-2 Diabetic Leprdb/Db Mice Show a Defective Microvascular Phenotype Under Basal Conditions and an Impaired Response to Angiogenesis Gene Therapy in the Setting of Limb Ischemia

Frontiers in Bioscience - Landmark - United States

doi 10.2741/2205

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Figure 3: Rosiglitazone Administration Increased Wound Angiogenesis in Db/Db Diabetic Mice.

Supplemental Information 3: Rosiglitazone Administration Increased Wound Angiogenesis in Db/Db Diabetic Mice

Non-Peptidyl Small Molecule, Adenosine, 5′-Se-Methyl-5′-Seleno-, 2′,3′-Diacetate, Activates Insulin Receptor and Attenuates Hyperglycemia in Type 2 Diabetic Leprdb/Db Mice

Intercellular Adhesion Molecule-1 Deficiency Is Protective Against Nephropathy in Type 2 Diabetic Db/Db Mice

Figure 2: Rosiglitazone Administration Accelerated Wound Closure in Db/Db Diabetic Mice.

Impaired Muscle Regeneration in Ob/Ob and Db/Db Mice

Ciliary Neurotrophic Factor Improves Diabetic Parameters and Hepatic Steatosis and Increases Basal Metabolic Rate in Db/Db Mice

Reduced Hyperaemia Following Skin Trauma: Evidence for an Impaired Microvascular Response to Injury in the Diabetic Foot

Divergent Systemic and Local Inflammatory Response to Hind Limb Demand Ischemia in Wild-Type and ApoE–/– Mice