(PDF) Comparison of the Effects of Insulin, PDGF,

Comparison of the Effects of Insulin, PDGF, Interleukin-6, and Interferon-G on Glucose Transport in 3t3-L1 Cells: Lack of Cross-Talk Between Tyrosine Kinase Receptors and JAK/STAT Pathways

Diabetologia - Germany

doi 10.1007/s001250050595

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November 21, 1996

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Springer Science and Business Media LLC

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Comparison of the Effects of Insulin, PDGF, Interleukin-6, and Interferon-G on Glucose Transport in 3t3-L1 Cells: Lack of Cross-Talk Between Tyrosine Kinase Receptors and JAK/STAT Pathways

Diabetologia - Germany

doi 10.1007/s001250050595

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Cross-Talk Between the Platelet-Derived Growth Factor and the Insulin Signaling Pathways in 3t3-L1 Adipocytes

Cross-Talk Between Janus Kinase-Signal Transducer and Activator of Transcription (JAK-STAT) and Peroxisome Proliferator-Activated Receptor-Α (PPARα) Signaling Pathways

Involvement of PI 3-Kinase in Stimulation of Glucose Transport and Recruitment of Transferrin Receptors in 3t3-L1 Adipocytes

Mutation in the Jak Kinase JH2 Domain Hyperactivates Drosophila and Mammalian Jak-Stat Pathways.

Developmentally Spliced PKCβII Provides a Possible Link Between mTORC2 and Akt Kinase to Regulate 3t3-L1 Adipocyte Insulin-Stimulated Glucose Transport

Kinetic Resolution of the Separate GLUT1 and GLUT4 Glucose Transport Activities in 3t3-L1 Cells

Activation of Protein Kinase a Inhibits Interferon Induction of the Jak/Stat Pathway in U266 Cells

Targeting the Interleukin-6/Jak/Stat Pathway in Human Malignancies

Improvement of High-Glucose and Insulin Resistance of Chromium Malate in 3t3-L1 Adipocytes by Glucose Uptake and Insulin Sensitivity Signaling Pathways and Its Mechanism