(PDF) Leucettine L41, a DYRK1A-preferential DYRKs/CLKs

Leucettine L41, a DYRK1A-preferential DYRKs/CLKs Inhibitor, Prevents Memory Impairments and Neurotoxicity Induced by Oligomeric Aβ25–35 Peptide Administration in Mice

European Neuropsychopharmacology - Netherlands

doi 10.1016/j.euroneuro.2015.03.018

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November 1, 2015

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Elsevier BV

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Leucettine L41, a DYRK1A-preferential DYRKs/CLKs Inhibitor, Prevents Memory Impairments and Neurotoxicity Induced by Oligomeric Aβ25–35 Peptide Administration in Mice

European Neuropsychopharmacology - Netherlands

doi 10.1016/j.euroneuro.2015.03.018

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

MGCD0103, a Selective Histone Deacetylase Inhibitor, Coameliorates Oligomeric Aβ25-35 -Induced Anxiety and Cognitive Deficits in a Mouse Model

Compound Danshen Tablet Ameliorated Aβ25-35-Induced Spatial Memory Impairment in Mice via Rescuing Imbalance Between Cytokines and Neurotrophins

Neuroprotection by the Synthetic Neurosteroid Enantiomers Ent-Pregs and Ent-Dheas Against Aβ25–35 Peptide-Induced Toxicity in Vitro and in Vivo in Mice

Endophilin 1 Knockdown Prevents Synaptic Dysfunction Induced by Oligomeric Amyloid Β

Exercise Prevents Memory Impairment Induced by Arsenic Exposure in Mice: Implication of Hippocampal BDNF and CREB

JAK2/STAT5/Bcl-xL Signalling Is Essential for Erythropoietin-Mediated Protection Against Apoptosis Induced in PC12 Cells by the Amyloid Β−peptide Aβ25-35

PPARγ Activation Prevents Impairments in Spatial Memory and Neurogenesis Following Transient Illness

Manipulation of Alternative Splicing by a Newly Developed Inhibitor of CLKS

Asymptomatic Neurotoxicity of Amyloid Β-Peptides (Aβ1-42 and Aβ25-35) on Mouse Embryonic Stem Cell-Derived Neural Cells