Enhanced Glycemic Responsiveness to Epinephrine in Insulin-Dependent Diabetes Mellitus Is the Result of the Inability to Secrete Insulin. Augmented Insulin Secretion Normally Limits the Glycemic, but Not the Ipolytic or Ketogenic, Response to Epinephrine in Humans

Journal of Clinical Investigation - United States
doi 10.1172/jci112176c1
Full Text
Abstract

Available in full text

Categories
Medicine
Date
Authors

Unknown

Publisher

American Society for Clinical Investigation