Critical Role of the NADPH Oxidase Subunit P47phox on Vascular TLR Expression and Neointimal Lesion Formation in High-Fat Diet-Induced Obesity

Laboratory Investigation - United Kingdom
doi 10.1038/labinvest.2008.92
Full Text
Abstract

Available in full text

Date
Authors
Publisher

Springer Science and Business Media LLC

Related search

Activation of P47PHOX, a Cytosolic Subunit of the Leukocyte NADPH Oxidase

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
1998English

Deficiency in the NADPH Oxidase 4 Predisposes Towards Diet-Induced Obesity

International Journal of Obesity
NutritionEndocrinologyDieteticsMedicineMetabolismDiabetes
2012English

High-Fat Diet-Induced Obesity in Animal Models

Nutrition Research Reviews
MedicineNutritionDietetics
2010English

Vascular Effects Following Homozygous Disruption of P47phox : An Essential Component of NADPH Oxidase

Circulation
Cardiovascular MedicinePhysiologyCardiology
2000English

High Fat Diet Induced Obesity Alters Ovarian Phosphatidylinositol-3 Kinase Signaling Gene Expression

Reproductive Toxicology
Toxicology
2013English

GDF15 Deficiency Promotes High Fat Diet-Induced Obesity in Mice

PLoS ONE
Multidisciplinary
2018English

Role of Endothelin Receptor a and NADPH Oxidase in Vascular Abnormalities

Vascular Health and Risk Management
2010English

Specificity of P47phox SH3 Domain Interactions in NADPH Oxidase Assembly and Activation.

Molecular and Cellular Biology
Cell BiologyMolecular Biology
1997English

S100A4 Protects Mice From High-Fat Diet-Induced Obesity and Inflammation

Laboratory Investigation
Forensic MedicinePathologyCell BiologyMolecular Biology
2018English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy