(PDF) Cigarette Smoke Reduces Fatty Acid Catabolism,

Cigarette Smoke Reduces Fatty Acid Catabolism, Leading to Apoptosis in Lung Endothelial Cells: Implication for Pathogenesis of COPD

Frontiers in Pharmacology - Switzerland

doi 10.3389/fphar.2019.00941

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August 29, 2019

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Cigarette Smoke Reduces Fatty Acid Catabolism, Leading to Apoptosis in Lung Endothelial Cells: Implication for Pathogenesis of COPD

Frontiers in Pharmacology - Switzerland

doi 10.3389/fphar.2019.00941

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Serotonin and Corticosterone Rhythms in Mice Exposed to Cigarette Smoke and in Patients With COPD: Implication for COPD-Associated Neuropathogenesis

Cigarette Smoke Extract Reduces VEGF in Primary Human Airway Epithelial Cells

S146 Microvascular Endothelial Cell Apoptosis and Dysregulation of Vascular Endothelial Growth Factor Receptor 2 (VEGF-R2) in Response to Cigarette Smoke. New Insights Into the Pathogenesis of Emphysema

Cigarette Smoke Extract Induces Oxidative Stress and Apoptosis in Human Lung Fibroblasts

VEGFR-2 Inhibition Augments Cigarette Smoke-Induced Oxidative Stress and Inflammatory Responses Leading to Endothelial Dysfunction

14,15-Epoxyeicosatrienoic Acid Suppresses Cigarette Smoke Condensate-Induced Inflammation in Lung Epithelial Cells by Inhibiting Autophagy

Chronic Cigarette Smoke Causes Oxidative Damage and Apoptosis to Retinal Pigmented Epithelial Cells in Mice

Cigarette Smoke Impairs Endothelial Cell Prostacyclin Production

Trans-Resveratrol Reduces Cardiac Oxidative Stress in Rats Exposed to Cigarette Smoke