(PDF) Apigenin Blocks Lipopolysaccharide-Induced Lethality

Apigenin Blocks Lipopolysaccharide-Induced Lethality in Vivo and Proinflammatory Cytokines Expression by Inactivating NF- B Through the Suppression of P65 Phosphorylation

Journal of Immunology - United States

doi 10.4049/jimmunol.179.10.7121

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November 2, 2007

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The American Association of Immunologists

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Apigenin Blocks Lipopolysaccharide-Induced Lethality in Vivo and Proinflammatory Cytokines Expression by Inactivating NF- B Through the Suppression of P65 Phosphorylation

Journal of Immunology - United States

doi 10.4049/jimmunol.179.10.7121

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Suppression of NF-κB and NF-κB-Regulated Gene Expression by Apigenin Through IκBα and IKK Pathway in TRAMP Mice

SIRT2 Regulates NF- B-Dependent Gene Expression Through Deacetylation of P65 Lys310

Transient and Selective NF- B P65 Serine 536 Phosphorylation Induced by T Cell Costimulation Is Mediated by I B Kinase and Controls the Kinetics of P65 Nuclear Import

AKIP1 Enhances NF-κB-dependent Gene Expression by Promoting the Nuclear Retention and Phosphorylation of P65

Betulinic Acid Suppresses Carcinogen-Induced NF- B Activation Through Inhibition of I B Kinase and P65 Phosphorylation: Abrogation of Cyclooxygenase-2 and Matrix Metalloprotease-9

Antisense Inhibition of the P65 Subunit of NF-kappa B Blocks Tumorigenicity and Causes Tumor Regression.

Interleukin-6 Facilitates Lipopolysaccharide-Induced Disruption in Working Memory and Expression of Other Proinflammatory Cytokines in Hippocampal Neuronal Cell Layers

Nanog Inhibits Lipopolysaccharide-Induced Expression of Pro-Inflammatory Cytokines by Blocking NF-κB Transcriptional Activity in Rat Primary Microglial Cells

Suppression of Bank Vole Pancreatic Islet Function by Proinflammatory Cytokines