Evidence in Support of a Role for Human T-Cell Leukemia Virus Type I Tax in Activating NF-B via Stimulation of Signaling Pathways

Journal of Biological Chemistry- United States

doi 10.1074/jbc.270.20.11745

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Date

May 19, 1995

Authors

Tomohiko Kanno

Keith Brown

Ulrich Siebenlist

Publisher

American Society for Biochemistry & Molecular Biology (ASBMB)

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Evidence in Support of a Role for Human T-Cell Leukemia Virus Type I Tax in Activating NF-B via Stimulation of Signaling Pathways

Journal of Biological Chemistry- United States

Full Text

Abstract

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Date

Authors

Publisher

Related search

Kinetic Analysis of Human T-Cell Leukemia Virus Type I Tax-Mediated Activation of NF-kappa B.

Activation of NF- B by the Human T Cell Leukemia Virus Type I Tax Oncoprotein Is Associated With Ubiquitin-Dependent Relocalization of I B Kinase

Development of Leukemia in Mice Transgenic for the Tax Gene of Human T-Cell Leukemia Virus Type I.

A Human T-Cell Leukemia Virus Tax Variant Incapable of Activating NF-κB Retains Its Immortalizing Potential for Primary T-Lymphocytes

Human T-Cell Leukemia Virus Type I Tax Down-Regulates the Expression of Phosphatidylinositol 3,4,5-Trisphosphate Inositol Phosphatases via the NF-κB Pathway.

Genomic Instability Driven by the Human T-Cell Leukemia Virus Type I (HTLV-I) Oncoprotein, Tax

Constitutive Dephosphorylation and Activation of a Member of the Nuclear Factor of Activated T Cells, NF-AT1, in Tax-Expressing and Type I Human T-Cell Leukemia Virus-Infected Human T Cells

Human T-Cell Leukemia Virus Type I Tax Protein Transactivates RNA Polymerase III Promoterin Vitroandin Vivo

Human T Cell Lymphotropic Virus Type I Genomic Expression and Impact on Intracellular Signaling Pathways During Neurodegenerative Disease and Leukemia