(PDF) A Succession of Mechanisms Stimulate Efficient

A Succession of Mechanisms Stimulate Efficient Reconstituted HIV-1 Minus Strand Strong Stop DNA Transfer†

Biochemistry - United States

doi 10.1021/bi802149j

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March 3, 2009

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American Chemical Society (ACS)

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A Succession of Mechanisms Stimulate Efficient Reconstituted HIV-1 Minus Strand Strong Stop DNA Transfer†

Biochemistry - United States

doi 10.1021/bi802149j

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Plus-Strand Strong-Stop DNA Synthesis in Retrotransposon Ty1.

A Sequence Similar to tRNA3Lys Gene Is Embedded in HIV-1 U3–R and Promotes Minus-Strand Transfer

HIV-1 Integrase Strand Transfer Inhibitors Stabilize an Integrase–Single Blunt-Ended DNA Complex

Utilization of Nonviral Sequences for Minus-Strand DNA Transfer and Gene Reconstitution During Retroviral Replication

Single-Molecule FRET Studies of Important Intermediates in the Nucleocapsid-Protein-Chaperoned Minus-Strand Transfer Step in HIV-1 Reverse Transcription

Factors That Determine the Efficiency of HIV-1 Strand Transfer Initiated at a Specific Site

Cryo-Em Structures and Atomic Model of the HIV-1 Strand Transfer Complex Intasome

HIV-1 Nucleocapsid Protein Increases Strand Transfer Recombination by Promoting Dimeric G-Quartet Formation

Sequence Identity of the Terminal Redundancies on the Minus-Strand DNA Template Is Necessary but Not Sufficient for the Template Switch During Hepadnavirus Plus-Strand DNA Synthesis.