Formation of Advanced Glycation End-Product-Modified Superoxide Dismutase-1 (SOD1) Is One of the Mechanisms Responsible for Inclusions Common to Familial Amyotrophic Lateral Sclerosis Patients With SOD1 Gene Mutation, and Transgenic Mice Expressing Human SOD1 Gene Mutation

Neuropathology - United Kingdom

doi 10.1046/j.1440-1789.2001.00359.x