(PDF) High Glucose Inhibits the Aspirin-Induced Activation

High Glucose Inhibits the Aspirin-Induced Activation of the Nitric Oxide/cGMP/cGMP-Dependent Protein Kinase Pathway and Does Not Affect the Aspirin-Induced Inhibition of Thromboxane Synthesis in Human Platelets

Diabetes - United States

doi 10.2337/db12-0040

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July 26, 2012

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American Diabetes Association

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High Glucose Inhibits the Aspirin-Induced Activation of the Nitric Oxide/cGMP/cGMP-Dependent Protein Kinase Pathway and Does Not Affect the Aspirin-Induced Inhibition of Thromboxane Synthesis in Human Platelets

Diabetes - United States

doi 10.2337/db12-0040

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Mechanisms Associated With cGMP Binding and Activation of cGMP-dependent Protein Kinase

Aspirin Inhibits LPS-induced Macrophage Activation via the NF-κB Pathway

Activation of P38 Mitogen-Activated Protein Kinase by Lipopolysaccharide in Human Neutrophils Requires Nitric Oxide-Dependent cGMP Accumulation

Nitrosyl-Cobinamide (NO-Cbi), a New Nitric Oxide Donor, Improves Wound Healing Through cGMP/cGMP-dependent Protein Kinase

Nitric Oxide Protects PC12 Cells From Serum Deprivation-Induced Apoptosis by cGMP-Dependent Inhibition of Caspase Signaling

Nitric Oxide Depresses GABAAReceptor Function via Coactivation of cGMP-Dependent Kinase and Phosphodiesterase

cGMP-Dependent Protein Kinase in Dorsal Root Ganglion: Relationship With Nitric Oxide Synthase and Nociceptive Neurons

Quinone-Induced Activation of Keap1/Nrf2 Signaling by Aspirin Prodrugs Masquerading as Nitric Oxide

Nitric Oxide-Donating Aspirin Inhibits the Growth of Pancreatic Cancer Cells Through Redox-Dependent Signaling