(PDF) Poxvirus Protein N1L Targets the I-κB Kinase Complex,

Poxvirus Protein N1L Targets the I-κB Kinase Complex, Inhibits Signaling to NF-κB by the Tumor Necrosis Factor Superfamily of Receptors, and Inhibits NF-κB and IRF3 Signaling by Toll-Like Receptors

Journal of Biological Chemistry - United States

doi 10.1074/jbc.m400567200

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Date

June 23, 2004

Authors

Katherine A. Fitzgerald

William L. Marshall

Publisher

American Society for Biochemistry & Molecular Biology (ASBMB)

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Poxvirus Protein N1L Targets the I-κB Kinase Complex, Inhibits Signaling to NF-κB by the Tumor Necrosis Factor Superfamily of Receptors, and Inhibits NF-κB and IRF3 Signaling by Toll-Like Receptors

Journal of Biological Chemistry - United States

doi 10.1074/jbc.m400567200

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Curcumin Inhibits McF‑7 Cells by Modulating the NF‑κB Signaling Pathway

Tumor Necrosis Factor-Like Weak Inducer of Apoptosis (TWEAK) Promotes Glioma Cell Invasion Through Induction of NF-κB-inducing Kinase (NIK) and Noncanonical NF-κB Signaling

IRF3 Negatively Regulates Toll-Like Receptor-Mediated NF-κB Signaling by Targeting TRIF for Degradation in Teleost Fish

Fas-Associated Factor-1 Inhibits Nuclear Factor-κB (NF-κB) Activity by Interfering With Nuclear Translocation of the RelA (P65) Subunit of NF-κB

Heat Shock Inhibits Radiation-Induced Activation of NF-κB via Inhibition of I-κB Kinase

Tussilagone Inhibits Osteoclastogenesis and Periprosthetic Osteolysis by Suppressing the NF-κB and P38 MAPK Signaling Pathways

Mumps Virus SH Protein Inhibits NF-κB Activation by Interacting With Tumor Necrosis Factor Receptor 1, Interleukin-1 Receptor 1, and Toll-Like Receptor 3 Complexes

Ubiquitin in NF-κB Signaling

CD52 Inhibits Toll-Like Receptor Activation of NF-κB and Triggers Apoptosis to Suppress Inflammation