Silencing Kinase-Interacting Stathmin Gene Enhances Erlotinib Sensitivity by Inhibiting Ser10 P27 Phosphorylation in Epidermal Growth Factor Receptor-Expressing Breast Cancer

Molecular Cancer Therapeutics - United States
doi 10.1158/1535-7163.mct-10-0362
Full Text
Abstract

Available in full text

Date
Authors
Publisher

American Association for Cancer Research (AACR)

Related search

Continuous Inhibition of Epidermal Growth Factor Receptor Phosphorylation by Erlotinib Enhances Antitumor Activity of Chemotherapy in Erlotinib-Resistant Tumor Xenografts

Oncology Reports
MedicineCancer ResearchOncology
2011English

Nuclear Translocation of Epidermal Growth Factor Receptor by Akt-Dependent Phosphorylation Enhances Breast Cancer-Resistant Protein Expression in Gefitinib-Resistant Cells

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
2011English

Role of Epidermal Growth Factor Receptor in Breast Cancer

Breast Cancer Research and Treatment
Cancer ResearchOncology
2012English

Herceptin Enhances the Antitumor Effect of Natural Killer Cells on Breast Cancer Cells Expressing Human Epidermal Growth Factor Receptor-2

Frontiers in Immunology
AllergyImmunology
2017English

Pgrmc1 (Progesterone Receptor Membrane Component 1) Associates With Epidermal Growth Factor Receptor and Regulates Erlotinib Sensitivity

Journal of Biological Chemistry
BiochemistryCell BiologyMolecular Biology
2010English

Human Epidermal Growth Factor Receptor 2-Targeted Therapies in Breast Cancer

Expert Opinion on Biological Therapy
Drug DiscoveryClinical BiochemistryPharmacology
2013English

Epidermal Growth Factor Receptor in Breast Cancer. Comparison With Non Malignant Breast Tissue

British Journal of Cancer
Cancer ResearchOncology
1993English

Heparin-Binding Epidermal Growth Factor Cleavage Mediates Zinc-Induced Epidermal Growth Factor Receptor Phosphorylation

American Journal of Respiratory Cell and Molecular Biology
Respiratory MedicinePulmonaryClinical BiochemistryCell BiologyMolecular Biology
2004English

Activation of C-Fos Gene Expression by a Kinase-Deficient Epidermal Growth Factor Receptor.

Molecular and Cellular Biology
Cell BiologyMolecular Biology
1994English

Amanote Research

Note-taking for researchers

Follow Amanote

© 2024 Amaplex Software S.P.R.L. All rights reserved.

Privacy PolicyRefund Policy