Amanote Research
Register
Sign In
PPARγ Regulated CIDEA Affects Pro-Apoptotic Responses in Glioblastoma
Cell Death Discovery
- United Kingdom
doi 10.1038/cddiscovery.2015.38
Full Text
Open PDF
Abstract
Available in
full text
Categories
Cancer Research
Molecular Neuroscience
Immunology
Cell Biology
Cellular
Date
November 23, 2015
Authors
A Chatterjee
P Mondal
S Ghosh
VS Mehta
E Sen
Publisher
Springer Science and Business Media LLC
Related search
Apoptotic Signaling Pathways in Glioblastoma and Therapeutic Implications
BioMed Research International
Immunology
Molecular Biology
Biochemistry
Microbiology
Medicine
Genetics
Involvement of Pro-Apoptotic and Pro-Autophagic Proteins in Granulosa Cell Death
Cell Biology
Activation of Pro-Apoptotic Caspases in Non-Apoptotic Cells During Odontogenesis and Related Osteogenesis
Frontiers in Physiology
Physiology
Quantitative PPARγ Expression Affects the Balance Between Tolerance and Immunity
Scientific Reports
Multidisciplinary
Expression of Bbc3, a Pro-Apoptotic BH3-only Gene, Is Regulated by Diverse Cell Death and Survival Signals
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
The Development of Pro-Apoptotic Cancer Therapeutics
Mini-Reviews in Medicinal Chemistry
Medicine
Drug Discovery
Pharmacology
Mapping the Interaction of Pro-Apoptotic tBID With Pro-Survival BCL-XL
Biochemistry
Biochemistry
Pro-Inflammatory Gene Expression in Solid Glioblastoma Microenvironment and in Hypoxic Stem Cells From Human Glioblastoma
Journal of Neuroinflammation
Molecular Neuroscience
Neurology
Immunology
Cellular
Neuroscience
Mitochondrial Apoptotic Priming Regulates Therapeutic Responses in AML
Cancer Discovery
Oncology