(PDF) Betulinic Acid Suppresses Carcinogen-Induced NF- B

Betulinic Acid Suppresses Carcinogen-Induced NF- B Activation Through Inhibition of I B Kinase and P65 Phosphorylation: Abrogation of Cyclooxygenase-2 and Matrix Metalloprotease-9

Journal of Immunology - United States

doi 10.4049/jimmunol.171.6.3278

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Date

September 5, 2003

Authors

Yasunari Takada

Bharat B. Aggarwal

Publisher

The American Association of Immunologists

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Amanote Research

Betulinic Acid Suppresses Carcinogen-Induced NF- B Activation Through Inhibition of I B Kinase and P65 Phosphorylation: Abrogation of Cyclooxygenase-2 and Matrix Metalloprotease-9

Journal of Immunology - United States

doi 10.4049/jimmunol.171.6.3278

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Transient and Selective NF- B P65 Serine 536 Phosphorylation Induced by T Cell Costimulation Is Mediated by I B Kinase and Controls the Kinetics of P65 Nuclear Import

Deguelin, an Akt Inhibitor, Suppresses I B Kinase Activation Leading to Suppression of NF- B-Regulated Gene Expression, Potentiation of Apoptosis, and Inhibition of Cellular Invasion

-Arrestin 2 Is Required for Lysophosphatidic Acid-Induced NF- B Activation

Embelin Suppresses Osteoclastogenesis Induced by Receptor Activator of NF- B Ligand and Tumor Cells in Vitro Through Inhibition of the NF- B Cell Signaling Pathway

Apigenin Blocks Lipopolysaccharide-Induced Lethality in Vivo and Proinflammatory Cytokines Expression by Inactivating NF- B Through the Suppression of P65 Phosphorylation

Cardiomyocyte-Specific I B Kinase (IKK)/NF- B Activation Induces Reversible Inflammatory Cardiomyopathy and Heart Failure

SIRT2 Regulates NF- B-Dependent Gene Expression Through Deacetylation of P65 Lys310

A Key Role for Mast Cell Chymase in the Activation of Pro-Matrix Metalloprotease-9 and Pro-Matrix Metalloprotease-2

Antisense Inhibition of the P65 Subunit of NF-kappa B Blocks Tumorigenicity and Causes Tumor Regression.