Noncanonical NF-κB Signaling and the Essential Kinase NIK Modulate Crucial Features Associated With Eosinophilic Esophagitis Pathogenesis

DMM Disease Models and Mechanisms- United Kingdom

doi 10.1242/dmm.030767

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December 1, 2017

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The Company of Biologists

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Noncanonical NF-κB Signaling and the Essential Kinase NIK Modulate Crucial Features Associated With Eosinophilic Esophagitis Pathogenesis

DMM Disease Models and Mechanisms- United Kingdom

Full Text

Abstract

Categories

Date

Authors

Publisher

Related search

Tumor Necrosis Factor-Like Weak Inducer of Apoptosis (TWEAK) Promotes Glioma Cell Invasion Through Induction of NF-κB-inducing Kinase (NIK) and Noncanonical NF-κB Signaling

The Kinase TBK1 Controls IgA Class Switching by Negatively Regulating Noncanonical NF-κB Signaling

Activation of Noncanonical NF-κB Signaling by the Oncoprotein Tio

Noncanonical NF-κB Activation Requires Coordinated Assembly of a Regulatory Complex of the Adaptors cIAP1, cIAP2, TRAF2 and TRAF3 and the Kinase NIK

Eosinophilic Esophagitis: Novel Concepts Regarding Pathogenesis and Clinical Manifestations

Inhibition of NF-κB Signaling Retards Eosinophilic Dermatitis in SHARPIN-Deficient Mice

Local and Systemic IKKεand NF-κB Signaling Associated With Sjögren’s Syndrome Immunopathogenesis

Expression of Intracellular Components of the NF-κB Alternative Pathway (NF-κB2, RelB, NIK and Bcl3) Is Associated With Clinical Outcome of NSCLC Patients

Eosinophilic Esophagitis Reference Score Can Identify Pediatric Patients With Eosinophilic Esophagitis