Noncanonical NF-κB Activation Requires Coordinated Assembly of a Regulatory Complex of the Adaptors cIAP1, cIAP2, TRAF2 and TRAF3 and the Kinase NIK

Nature Immunology- United Kingdom

doi 10.1038/ni.1676

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November 9, 2008

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Springer Science and Business Media LLC

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Noncanonical NF-κB Activation Requires Coordinated Assembly of a Regulatory Complex of the Adaptors cIAP1, cIAP2, TRAF2 and TRAF3 and the Kinase NIK

Nature Immunology- United Kingdom

Full Text

Abstract

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Date

Authors

Publisher

Related search

Noncanonical NF-κB Signaling and the Essential Kinase NIK Modulate Crucial Features Associated With Eosinophilic Esophagitis Pathogenesis

Tumor Necrosis Factor-Like Weak Inducer of Apoptosis (TWEAK) Promotes Glioma Cell Invasion Through Induction of NF-κB-inducing Kinase (NIK) and Noncanonical NF-κB Signaling

Activation of Noncanonical NF-κB Signaling by the Oncoprotein Tio

Conditional Deletion of NF-κB–Inducing Kinase (NIK) in Adult Mice Disrupts Mature B Cell Survival and Activation

NIK–IKK Complex Interaction Controls NF-κB-dependent Inflammatory Activation of Endothelium in Response to LTβR Ligation

The IκB Kinase Complex in NF-κB Regulation and Beyond

The Kinase TBK1 Controls IgA Class Switching by Negatively Regulating Noncanonical NF-κB Signaling

Regulation of the NF-κB Activation Pathway by Isolated Domains of FIP3/IKKγ, a Component of the IκB-α Kinase Complex

Beyond NF-κB Activation: Nuclear Functions of IκB Kinase Α