Hypocretin and Brain Î²-Amyloid Peptide Interactions in Cognitive Disorders and Narcolepsy

Frontiers in Aging Neuroscience- Switzerland

doi 10.3389/fnagi.2014.00119

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June 11, 2014

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Frontiers Media SA

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Hypocretin and Brain Î²-Amyloid Peptide Interactions in Cognitive Disorders and Narcolepsy

Frontiers in Aging Neuroscience- Switzerland

Full Text

Abstract

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Publisher

Related search

0016 Autoimmunity to Hypocretin and Molecular Mimicry to Flu in Type 1 Narcolepsy

Opiates Increase the Number of Hypocretin-Producing Cells in Human and Mouse Brain and Reverse Cataplexy in a Mouse Model of Narcolepsy

Roles of Amyloid Β-Peptide-Associated Oxidative Stress and Brain Protein Modifications in the Pathogenesis of Alzheimer's Disease and Mild Cognitive Impairment

Hypocretin Deficiency Associated With Narcolepsy Type 1 and Central Hypoventilation Syndrome in Neurosarcoidosis of the Hypothalamus

Cross-Interactions Between the Alzheimer Disease Amyloid-Β Peptide and Other Amyloid proteins.A FURTHER ASPECT OF THE AMYLOID CASCADE HYPOTHESIS.

Gene-Environment Interactions: Lifetime Cognitive Activity, APOE Genotype, and Beta-Amyloid Burden

Sesaminol Glucosides Protect Β-Amyloid Peptide-Induced Cognitive Deficits in Mice

Direct and Indirect Excitation of Laterodorsal Tegmental Neurons by Hypocretin/Orexin Peptides: Implications for Wakefulness and Narcolepsy

Genetic Reductions of Î²-Site Amyloid Precursor Protein-Cleaving Enzymeâ1 and Amyloid-Î² Ameliorate Impairment of Conditioned Taste Aversion Memory in 5XFAD Alzheimerâs Disease Model Mice

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Hypocretin and Brain Î²-Amyloid Peptide Interactions in Cognitive Disorders and Narcolepsy

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Full Text

Abstract

Categories

Date

Authors

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Related search

0016 Autoimmunity to Hypocretin and Molecular Mimicry to Flu in Type 1 Narcolepsy

Opiates Increase the Number of Hypocretin-Producing Cells in Human and Mouse Brain and Reverse Cataplexy in a Mouse Model of Narcolepsy

Roles of Amyloid Β-Peptide-Associated Oxidative Stress and Brain Protein Modifications in the Pathogenesis of Alzheimer's Disease and Mild Cognitive Impairment

Hypocretin Deficiency Associated With Narcolepsy Type 1 and Central Hypoventilation Syndrome in Neurosarcoidosis of the Hypothalamus

Cross-Interactions Between the Alzheimer Disease Amyloid-Β Peptide and Other Amyloid proteins.A FURTHER ASPECT OF THE AMYLOID CASCADE HYPOTHESIS.

Gene-Environment Interactions: Lifetime Cognitive Activity, APOE Genotype, and Beta-Amyloid Burden

Sesaminol Glucosides Protect Β-Amyloid Peptide-Induced Cognitive Deficits in Mice

Direct and Indirect Excitation of Laterodorsal Tegmental Neurons by Hypocretin/Orexin Peptides: Implications for Wakefulness and Narcolepsy

Genetic Reductions of Î²-Site Amyloid Precursor Protein-Cleaving Enzymeâ1 and Amyloid-Î² Ameliorate Impairment of Conditioned Taste Aversion Memory in 5XFAD Alzheimerâs Disease Model Mice

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