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Publications by D. Gasparro

Early-Onset Type 2 Diabetes in Obese White Subjects Is Characterised by a Marked Defect in Beta Cell Insulin Secretion, Severe Insulin Resistance and a Lack of Response to Aerobic Exercise Training

Diabetologia

English

Related publications

Impairment of Early Insulin Response After Glucose Load, Rather Than Insulin Resistance, Is Responsible for Postprandial Hyperglycemia Seen in Obese Type 2 Diabetes. Assessment Using Nateglinide, a New Insulin Secretagogue.

English

Vinegar Improves Insulin Sensitivity to a High-Carbohydrate Meal in Subjects With Insulin Resistance or Type 2 Diabetes

English

Different Aetiologies of Type 2 (Non-Insulin-Dependent) Diabetes Mellitus in Obese and Non-Obese Subjects

English

Changes in Plasma Visfatin and Insulin Resistance Index in Obese Women With Type 2 Diabetes After Pilates Exercise

Journal of Health and Care

2018

English

PCLO Variants Are Nominally Associated With Early-Onset Type 2 Diabetes and Insulin Resistance in Pima Indians

English

Acute Lowering of Circulating Fatty Acids Improves Insulin Secretion in a Subset of Type 2 Diabetes Subjects

American Journal of Physiology - Endocrinology and Metabolism

English

Cell Type–specific Immune Phenotypes Predict Loss of Insulin Secretion in New-Onset Type 1 Diabetes

JCI insight

Medicine

2019

English

SIRT5 Regulates Pancreatic Β‑cell Proliferation and Insulin Secretion in Type 2 Diabetes

Experimental and Therapeutic Medicine

English

HOMA-beta in the UKPDS and ADOPT. Is the Natural History of Type 2 Diabetes Characterised by a Progressive and Inexorable Loss of Insulin Secretory Function? Maybe? Maybe Not?

Diabetes and Vascular Disease Research

Internal Medicine

Cardiology

Endocrinology

Cardiovascular Medicine

Metabolism

Diabetes

2009

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by D. Gasparro

Early-Onset Type 2 Diabetes in Obese White Subjects Is Characterised by a Marked Defect in Beta Cell Insulin Secretion, Severe Insulin Resistance and a Lack of Response to Aerobic Exercise Training

Related publications

Impairment of Early Insulin Response After Glucose Load, Rather Than Insulin Resistance, Is Responsible for Postprandial Hyperglycemia Seen in Obese Type 2 Diabetes. Assessment Using Nateglinide, a New Insulin Secretagogue.

Vinegar Improves Insulin Sensitivity to a High-Carbohydrate Meal in Subjects With Insulin Resistance or Type 2 Diabetes

Different Aetiologies of Type 2 (Non-Insulin-Dependent) Diabetes Mellitus in Obese and Non-Obese Subjects

Changes in Plasma Visfatin and Insulin Resistance Index in Obese Women With Type 2 Diabetes After Pilates Exercise

PCLO Variants Are Nominally Associated With Early-Onset Type 2 Diabetes and Insulin Resistance in Pima Indians

Acute Lowering of Circulating Fatty Acids Improves Insulin Secretion in a Subset of Type 2 Diabetes Subjects

Cell Type–specific Immune Phenotypes Predict Loss of Insulin Secretion in New-Onset Type 1 Diabetes

SIRT5 Regulates Pancreatic Β‑cell Proliferation and Insulin Secretion in Type 2 Diabetes

HOMA-beta in the UKPDS and ADOPT. Is the Natural History of Type 2 Diabetes Characterised by a Progressive and Inexorable Loss of Insulin Secretory Function? Maybe? Maybe Not?