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Publications by Emanuela Laratta

Angiotensin II Impairs the Insulin Signaling Pathway Promoting Production of Nitric Oxide by Inducing Phosphorylation of Insulin Receptor Substrate-1 on Ser312 and Ser616 in Human Umbilical Vein Endothelial Cells

Circulation Research

Cardiovascular Medicine

Physiology

Cardiology

2004

English

Related publications

Roles for Insulin Receptor, PI3-Kinase, and Akt in Insulin-Signaling Pathways Related to Production of Nitric Oxide in Human Vascular Endothelial Cells

Circulation

Cardiovascular Medicine

Physiology

Cardiology

2000

English

A Role for Heterotrimeric GTP-binding Proteins and ERK1/2 in Insulin-Mediated, Nitric-Oxide-Dependent, Cyclic GMP Production in Human Umbilical Vein Endothelial Cells

English

Inducible Nitric-Oxide Synthase and Nitric Oxide Donor Decrease Insulin Receptor Substrate-2 Protein Expression by Promoting Proteasome-Dependent Degradation in Pancreatic Β-Cells

Journal of Biological Chemistry

English

Effect of Interleukin-10 and Laminar Shear Stress on Endothelial Nitric Oxide Synthase and Nitric Oxide in African American Human Umbilical Vein Endothelial Cells

Ethnicity and Disease

Medicine

Epidemiology

2015

English

Insulin Stimulates PKCζ-mediated Phosphorylation of Insulin Receptor Substrate-1 (IRS-1)

Journal of Biological Chemistry

English

Piper Sarmentosum Promotes Endothelial Nitric Oxide Production by Reducing Asymmetric Dimethylarginine in Tumor Necrosis Factor-Α-Induced Human Umbilical Vein Endothelial Cells

Frontiers in Pharmacology

Pharmacology

2019

English

Regulation of Nitric Oxide Synthesis by Proinflammatory Cytokines in Human Umbilical Vein Endothelial Cells. Elevations in Tetrahydrobiopterin Levels Enhance Endothelial Nitric Oxide Synthase Specific Activity.

Journal of Clinical Investigation

Medicine

1994

English

Modulation of Insulin Receptor Substrate-1 Tyrosine Phosphorylation by an Akt/Phosphatidylinositol 3-Kinase Pathway

Journal of Biological Chemistry

English

Sphingomyelinase and Ceramide Suppress Insulin-Induced Tyrosine Phosphorylation of the Insulin Receptor Substrate-1

Journal of Biological Chemistry

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Emanuela Laratta

Angiotensin II Impairs the Insulin Signaling Pathway Promoting Production of Nitric Oxide by Inducing Phosphorylation of Insulin Receptor Substrate-1 on Ser312 and Ser616 in Human Umbilical Vein Endothelial Cells

Related publications

Roles for Insulin Receptor, PI3-Kinase, and Akt in Insulin-Signaling Pathways Related to Production of Nitric Oxide in Human Vascular Endothelial Cells

A Role for Heterotrimeric GTP-binding Proteins and ERK1/2 in Insulin-Mediated, Nitric-Oxide-Dependent, Cyclic GMP Production in Human Umbilical Vein Endothelial Cells

Inducible Nitric-Oxide Synthase and Nitric Oxide Donor Decrease Insulin Receptor Substrate-2 Protein Expression by Promoting Proteasome-Dependent Degradation in Pancreatic Β-Cells

Effect of Interleukin-10 and Laminar Shear Stress on Endothelial Nitric Oxide Synthase and Nitric Oxide in African American Human Umbilical Vein Endothelial Cells

Insulin Stimulates PKCζ-mediated Phosphorylation of Insulin Receptor Substrate-1 (IRS-1)

Piper Sarmentosum Promotes Endothelial Nitric Oxide Production by Reducing Asymmetric Dimethylarginine in Tumor Necrosis Factor-Α-Induced Human Umbilical Vein Endothelial Cells

Regulation of Nitric Oxide Synthesis by Proinflammatory Cytokines in Human Umbilical Vein Endothelial Cells. Elevations in Tetrahydrobiopterin Levels Enhance Endothelial Nitric Oxide Synthase Specific Activity.

Modulation of Insulin Receptor Substrate-1 Tyrosine Phosphorylation by an Akt/Phosphatidylinositol 3-Kinase Pathway

Sphingomyelinase and Ceramide Suppress Insulin-Induced Tyrosine Phosphorylation of the Insulin Receptor Substrate-1