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Publications by Junpei Mutoh
The Plant Flavonoid, Quercetin, Reduces Some Forms of Dioxin Toxicity by Mechanism Distinct From Aryl Hydrocarbon Receptor Activation, Heat-Shock Protein Induction and Quenching Oxidative Stress
JOURNAL OF HEALTH SCIENCE
Related publications
Aryl Hydrocarbon Receptor Activation by cAMP vs. Dioxin: Divergent Signaling Pathways
Proceedings of the National Academy of Sciences of the United States of America
Multidisciplinary
Role of Aryl Hydrocarbon Receptor-Mediated Induction of the CYP1 Enzymes in Environmental Toxicity and Cancer
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Role of Mediator in Transcriptional Activation by the Aryl Hydrocarbon Receptor
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Heat Shock and Oxidative Stress-Induced Exposure of Hydrophobic Protein Domains as Common Signal in the Induction Ofhsp68
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Induction of Colonic Regulatory T Cells by Mesalamine by Activating the Aryl Hydrocarbon Receptor
Cellular and Molecular Gastroenterology and Hepatology
Hepatology
Gastroenterology
Aryl Hydrocarbon Receptor Activation by Dioxin Targets Phosphoenolpyruvate Carboxykinase (PEPCK) for ADP-ribosylation via 2,3,7,8-Tetrachlorodibenzo-P-Dioxin (TCDD)-inducible Poly(ADP-Ribose) Polymerase (TiPARP)
Journal of Biological Chemistry
Biochemistry
Cell Biology
Molecular Biology
Polychlorinated Diphenylsulfides Activate Aryl Hydrocarbon Receptor 2 in Zebrafish Embryos: Potential Mechanism of Developmental Toxicity
2,3,7,8-Tetrachlorodibenzo-P-Dioxin-Induced Aryl Hydrocarbon Receptor Activation Enhanced the Suppressive Function of Mesenchymal Stem Cells Against Splenocyte Proliferation
In Vitro Cellular and Developmental Biology - Animal
Medicine
Developmental Biology
Cell Biology
Structural Basis for Aryl Hydrocarbon Receptor Mediated Gene Activation
