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Publications by Kathleen G. Dickman

Akt and C-Myc Differentially Activate Cellular Metabolic Programs and Prime Cells to Bioenergetic Inhibition

Journal of Biological Chemistry

English

Mice Lacking the VIP Gene Show Airway Hyperresponsiveness and Airway Inflammation, Partially Reversible by VIP

American Journal of Physiology - Lung Cellular and Molecular Physiology

English

Related publications

Human Pancreatic Cancer Cells Undergo Profound Metabolic Reprogramming Towards Cellular Stemness as Adaptation to Inhibition of the Akt Pathway

2020

English

Cellular Stresses Differentially Activate C-Jun N-Terminal Protein Kinases and Extracellular Signal-Regulated Protein Kinases in Cultured Ventricular Myocytes

Journal of Biological Chemistry

English

RNA Interference-Mediated C-Myc Inhibition Prevents Cell Growth and Decreases Sensitivity to Radio- And Chemotherapy in Childhood Medulloblastoma Cells

English

C-Myc Programs Fatty Acid Metabolism and Dictates Acetyl-CoA Abundance and Fate

Journal of Biological Chemistry

English

C-Myc Inhibition of MyoD and Myogenin-Initiated Myogenic Differentiation.

Molecular and Cellular Biology

Cell Biology

Molecular Biology

1991

English

Inhibition of C-Myc Expression Induces Apoptosis of WEHI 231 Murine B Cells.

Molecular and Cellular Biology

Cell Biology

Molecular Biology

1996

English

Human C-Myc Isoforms Differentially Regulate Cell Growth and Apoptosis in Drosophila Melanogaster

Molecular and Cellular Biology

Cell Biology

Molecular Biology

2005

English

An Epigenetic Pathway Regulates Sensitivity of Breast Cancer Cells to HER2 Inhibition via FOXO/c-Myc Axis

English

Myc Target in Myeloid Cells-1, a Novel C-Myc Target, Recapitulates Multiple C-Myc Phenotypes

Journal of Biological Chemistry

English

Amanote Research

Discover open access scientific publications

Search, annotate, share and cite publications

Publications by Kathleen G. Dickman

Akt and C-Myc Differentially Activate Cellular Metabolic Programs and Prime Cells to Bioenergetic Inhibition

Mice Lacking the VIP Gene Show Airway Hyperresponsiveness and Airway Inflammation, Partially Reversible by VIP

Related publications

Human Pancreatic Cancer Cells Undergo Profound Metabolic Reprogramming Towards Cellular Stemness as Adaptation to Inhibition of the Akt Pathway

Cellular Stresses Differentially Activate C-Jun N-Terminal Protein Kinases and Extracellular Signal-Regulated Protein Kinases in Cultured Ventricular Myocytes

RNA Interference-Mediated C-Myc Inhibition Prevents Cell Growth and Decreases Sensitivity to Radio- And Chemotherapy in Childhood Medulloblastoma Cells

C-Myc Programs Fatty Acid Metabolism and Dictates Acetyl-CoA Abundance and Fate

C-Myc Inhibition of MyoD and Myogenin-Initiated Myogenic Differentiation.

Inhibition of C-Myc Expression Induces Apoptosis of WEHI 231 Murine B Cells.

Human C-Myc Isoforms Differentially Regulate Cell Growth and Apoptosis in Drosophila Melanogaster

An Epigenetic Pathway Regulates Sensitivity of Breast Cancer Cells to HER2 Inhibition via FOXO/c-Myc Axis

Myc Target in Myeloid Cells-1, a Novel C-Myc Target, Recapitulates Multiple C-Myc Phenotypes